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Juvenile polyps occur in about 2 percent of children. [2] In contrast to other types of colon polyps, juvenile polyps are not premalignant and are not usually associated with a higher risk of cancer; [4] however, individuals with juvenile polyposis syndrome are at increased risk of gastric and colorectal cancer. [2] [5]
Juvenile polyposis syndrome is an autosomal dominant genetic condition characterized by the appearance of multiple juvenile polyps in the gastrointestinal tract. Polyps are abnormal growths arising from a mucous membrane .
Juvenile polyposis syndrome is characterised by the presence of more than five polyps in the colon or rectum, or numerous juvenile polyps throughout the gastrointestinal tract, or any number of juvenile polyps in any person with a family history of juvenile polyposis. People with juvenile polyposis have an increased risk of colon cancer. [19]
A polyp is an abnormal growth of tissue projecting from a mucous membrane. If it is attached to the surface by a narrow elongated stalk, it is said to be pedunculated; if it is attached without a stalk, it is said to be sessile. Polyps are commonly found in the colon, stomach, nose, ear, sinus(es), urinary bladder, and uterus.
Gardner's syndrome (also known as Gardner syndrome, familial polyposis of the colon, [1] or familial colorectal polyposis [2]) is a subtype of familial adenomatous polyposis (FAP). Gardner syndrome is an autosomal dominant form of polyposis characterized by the presence of multiple polyps in the colon together with tumors outside the colon. [ 3 ]
Cronkhite–Canada syndrome is a rare syndrome characterized by multiple polyps of the digestive tract.It is sporadic (i.e. it does not seem to be a hereditary disease), [1] and it is currently considered acquired [2] and idiopathic (i.e. cause remains unknown).
Particularly, exposure to the insecticide profenofos and the fungicide MEMC have played a major part in polyp retraction and biomass decrease. [10] [11] There have been many experiments supporting the hypothesis that heat stress in Acropora tenuis juvenile polyps provokes an up-regulation of protein in the endoplasmic reticulum. The results ...
Peutz–Jeghers syndrome (often abbreviated PJS) is an autosomal dominant genetic disorder characterized by the development of benign hamartomatous polyps in the gastrointestinal tract and hyperpigmented macules on the lips and oral mucosa (). [2]