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  2. Amyloid - Wikipedia

    en.wikipedia.org/wiki/Amyloid

    Amyloid shows up as homogeneous pink material in lamina propria and around blood vessels. 20× magnification. Amyloids are aggregates of proteins characterised by a fibrillar morphology of typically 7–13 nm in diameter, a β-sheet secondary structure (known as cross-β) and ability to be stained by particular dyes, such as Congo red. [1]

  3. Amyloid plaques - Wikipedia

    en.wikipedia.org/wiki/Amyloid_plaques

    Amyloid beta (Aβ) is a small protein, most often 40 or 42 amino acids in length, that is released from a longer parent protein called the Aβ-precursor protein (APP). [24] APP is produced by many types of cell in the body, but it is especially abundant in neurons. It is a single-pass transmembrane protein, passing once through cellular ...

  4. Amyloid (mycology) - Wikipedia

    en.wikipedia.org/wiki/Amyloid_(mycology)

    The term "amyloid" is derived from the Latin amyloideus ("starch-like"). [1] It refers to the fact that starch gives a similar reaction, also called an amyloid reaction. The test can be on microscopic features, such as spore walls or hyphal walls, or the apical apparatus or entire ascus wall of an ascus , or be a macroscopic reaction on tissue ...

  5. LECT2 amyloidosis - Wikipedia

    en.wikipedia.org/wiki/Lect2_amyloidosis

    LECT2 Amyloidosis (ALECT2) is a form of amyloidosis caused by the LECT2 protein. It was found to be the third most common (~3% of total) cause of amyloidosis in a set of more than 4,000 individuals studied at the Mayo Clinic; the first and second most common forms the disorder were AL amyloidosis and AA amyloidosis, respectively.

  6. APLP2 - Wikipedia

    en.wikipedia.org/wiki/APLP2

    Amyloid precursor like protein 2, also known as APLP2, is a protein encoded by the APLP2 gene in humans. [ 5 ] [ 6 ] APLP2 along with APLP1 are important modulators of glucose and insulin homeostasis .

  7. Hypocreomycetidae - Wikipedia

    en.wikipedia.org/wiki/Hypocreomycetidae

    The members of Hypocreomycetidae have light colored perithecia, non-amyloid or amyloid ascal rings, or those which lack apical rings and most taxa lack true paraphyses (Zhang et al. 2006). [3] Hypocreomycetidae was established by Eriksson and Winka (1997) based on morphology and a single gene phylogenetic analysis. [1]

  8. Alpha sheet - Wikipedia

    en.wikipedia.org/wiki/Alpha_sheet

    The alpha sheet has been proposed as a possible intermediate state in the conformational change in the formation of amyloid fibrils by peptides and proteins such as amyloid beta, poly-glutamine repeats, lysozyme, prion proteins, and transthyretin repeats, all of which are associated with protein misfolding disease.

  9. Serum amyloid P component - Wikipedia

    en.wikipedia.org/wiki/Serum_amyloid_P_component

    20219 Ensembl ENSG00000132703 ENSMUSG00000026542 UniProt P02743 P12246 RefSeq (mRNA) NM_001639 NM_011318 RefSeq (protein) NP_001630 NP_035448 Location (UCSC) Chr 1: 159.59 – 159.59 Mb Chr 1: 172.72 – 172.72 Mb PubMed search Wikidata View/Edit Human View/Edit Mouse The serum amyloid P component (SAP) is the identical serum form of the amyloid P component (AP), a 25 kDa pentameric protein ...