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A gluten-free diet (GFD) is a ... Coeliac disease (American English: celiac) (CD) ... and probably the reintroduction of both gluten and whey protein had a nocebo ...
For people with celiac disease, a lifelong strict gluten-free diet is the only effective treatment to date; [23] [69] For people diagnosed with non-celiac gluten sensitivity, there are still open questions concerning for example the duration of such a diet.
Coeliac disease (British English) or celiac disease (American English) is a long-term autoimmune disorder, primarily affecting the small intestine, where individuals develop intolerance to gluten, present in foods such as wheat, rye, spelt and barley. [10]
A part of people with gluten-related neuropathy or ataxia appears not to be able to tolerate even the traces of gluten allowed in most foods labeled as "gluten-free". [49] Whereas celiac disease requires adherence to a strict lifelong gluten-free diet, it is not yet known whether NCGS is a permanent or a transient condition.
Non-celiac gluten sensitivity (NCGS) is described as a condition of multiple symptoms that improves when switching to a gluten-free diet, after celiac disease and wheat allergy are excluded. [ 64 ] [ 65 ] Recognized since 2010, [ 66 ] [ 67 ] it is included among gluten-related disorders . [ 66 ]
This condition is known as refractory coeliac disease (RCD), defined as malabsorption due to gluten-related enteropathy (villous atrophy or elevated intraepitheal lymphocytes) after initial or subsequent failure of a strict gluten-free diet (usually 1 year) and after exclusion of any disorder mimicking coeliac disease. [106] [107]
The type of protein here is gluten, the main culprit in Celiac disease. Well, really the main culprit behind celiac disease is a 33 amino acid peptide component of gluten called gliadin. Okay, so the gliadin in gluten is what triggers immune attack in Celiac disease, hence the need for gluten-free pizza or buns in hamburgers, right?
The gluten sensitive gut: The fate of gluten in coeliac disease or EIA. There is a growing body of evidence that the gluten-sensitive intestine differs from the normal gut, several gluten peptides can enter behind the brush border membrane cells.
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