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Global aphasia is a severe form of nonfluent aphasia, caused by damage to the left side of the brain, that affects [1] receptive and expressive language skills (needed for both written and oral language) as well as auditory and visual comprehension. [2]
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In order to capitalize on neuroplasticity for treatment of all types of aphasia, timing, intensity, duration, and repetition of treatment should be taken into consideration. Research has found that aphasia treatment initiated during the earlier acute post-injury phase is more effective compared to treatment initiated in the chronic phase. [21]
Helm-Estabrooks is a researcher and speech-language pathologist (SLP) specializing in the design and application of widely used diagnostic. She developed, in collaboration with behavioral neurologist Martin Albert and Robert Sparks, a structured rehabilitation program for a type of nonfluent aphasia utilizing intoned phrases to facilitate speech and language production.
Transcortical sensory aphasia is characterized as a fluent aphasia. Fluency is determined by direct qualitative observation of the patient’s speech to determine the length of spoken phrases, and is usually characterized by a normal or rapid rate; normal phrase length, rhythm, melody, and articulatory agility; and normal or paragrammatic speech. [5]
Survivors with global aphasia may have great difficulty understanding and forming words and sentences, and generally experience a great deal of difficulty when trying to communicate. [2] With considerable speech therapy rehabilitation, global aphasia may progress into expressive aphasia or receptive aphasia.
Landau–Kleffner syndrome (LKS), also called infantile acquired aphasia, acquired epileptic aphasia, [1] or aphasia with convulsive disorder, is a rare neurological syndrome that develops during childhood. [2] It is named after William Landau and Frank Kleffner, who characterized it in 1957 with a diagnosis of six children. [3] [4]
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