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This contrasts with the pseudofusion beat wherein the pacemaker impulse does not affect the complex of the natural beat of the heart. Pseudofusion beats are normal. Rare or isolated fusion beats caused by pacemakers are normal as well, but if they occur too frequently may reduce cardiac output and so can require adjustment of the pacemaker. [3]
While the sinoatrial node typically regulates the heartbeat during normal sinus rhythm, PACs occur when another region of the atria depolarizes before the sinoatrial node and thus triggers a premature heartbeat, [1] in contrast to escape beats, in which the normal sinoatrial node fails, leaving a non-nodal pacemaker to initiate a late beat.
Accelerated junctional rhythm is when the rhythm of emerged impulses is more than 40 to 60 beats per minute, which is the natural range of the junction. It happens in some instances such as digoxin toxicity, and usually has a rate of between 60 and 100 bpm. [11]
Pacemaker syndrome is a condition that represents the clinical consequences of suboptimal atrioventricular (AV) synchrony or AV dyssynchrony, regardless of the pacing mode, after pacemaker implantation. [1] [2] It is an iatrogenic disease—an adverse effect resulting from medical treatment—that is often underdiagnosed.
Ectopic beat is a disturbance of the cardiac rhythm frequently related to the electrical conduction system of the heart, in which beats arise from fibers or group of fibers outside the region in the heart muscle ordinarily responsible for impulse formation (i.e., the sinoatrial node).
A premature heart beat or extrasystole [1] is a heart rhythm disorder corresponding to a premature contraction of one of the chambers of the heart. Premature heart beats come in two different types: premature atrial contractions and premature ventricular contractions. Often they cause no symptoms but may present with fluttering in the chest or ...
During ventricular activation, one bundle tract's area of slow conduction activates the other tract's bundle fibers post block after the rest of the ventricle has recovered. This resulting in an extra beat. Reentry can produce single ectopic beats, or it can trigger paroxysmal tachycardia.
During typical AVNRT, electrical impulses travel down the slow pathway of the AV node and back up the fast pathway. The fundamental mechanism of AVNRT is a presence of a dual atrioventricular node physiology (present in half of the population), which acts as a re-entrant circuit within the atrioventricular node. [4] This can take several forms.