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Nitrogen dioxide is an irritant of the mucous membrane linked with another air pollutant that causes pulmonary diseases such as obstructive lung disease, asthma, chronic obstructive pulmonary disease and sometimes acute exacerbation of COPD and in fatal cases, deaths. [2]
Combustion of gas can lead to increased concentrations of nitrogen dioxide throughout the home environment which is linked to respiratory issues and diseases. [9] [10] The LC 50 (median lethal dose) for humans has been estimated to be 174 ppm for a 1-hour exposure. [11] It is also included in the NO x family of atmospheric pollutants.
There is strong evidence that NO x respiratory exposure can trigger and exacerbate existing asthma symptoms, and may even lead to the development of asthma over longer periods of time. It has also been associated with heart disease, diabetes, birth outcomes, and all-cause mortality, but these nonrespiratory effects are less well-established. [29]
The role for eNO in other conditions is even less well established compared to asthma. Since asthma can be a cause of chronic coughing (it may even be the sole manifestation, such as in cough-variant asthma), studies have looked at whether eNO can be used in the diagnosis of chronic cough. [18] [19] [20] [21]
You’re also not alone, as more than 25 million Americans suffer from asthma. This chronic disease causes the lung’s airways to become inflamed and narrowed, making it difficult to breathe.
Air pollution can occur naturally or be caused by human activities. [4] Air pollution causes around 7 or 8 million deaths each year. [5] [6] It is a significant risk factor for a number of pollution-related diseases, including heart disease, stroke, chronic obstructive pulmonary disease (COPD), asthma and lung cancer.
Asthma phenotyping and endotyping has emerged as a novel approach to asthma classification inspired by precision medicine which separates the clinical presentations of asthma, or asthma phenotypes, from their underlying causes, or asthma endotypes. The best-supported endotypic distinction is the type 2-high/type 2-low distinction.
Occupational asthma is new onset asthma or the recurrence of previously quiescent asthma directly caused by exposure to an agent at workplace. It is an occupational lung disease and a type of work-related asthma. Agents that can induce occupational asthma can be grouped into sensitizers and irritants. [1]