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Debakey forceps, an "atraumatic" forceps used extensively in cardiothoracic, vascular and head and neck surgery. Thumb forceps, known simply as forceps in surgical specialties , are commonly held in a pen grip between the thumb and index finger (sometimes also the middle finger ), with the top end resting on the first dorsal interosseous muscle ...
The incidence of post-stroke depression peaks at 3–6 months and usually resolves within 1–2 years after the stroke, although a minority of patients can go on to develop chronic depression. The diagnosis of post-stroke depression is complicated by other consequences of stroke such as fatigue and psychomotor retardation – which do not ...
Debakey forceps. Debakey forceps are a type of atraumatic tissue forceps used in vascular procedures to avoid tissue damage during manipulation. They are typically large (some examples are upwards of 12 inches (36 cm) long), and have a distinct coarsely ribbed grip panel, as opposed to the finer ribbing on most other tissue forceps.
Used for tissue or object grasping. Forceps are categorized into toothed or non-toothed at the tip. [21] (e.g.,Tissue forceps, Adson forceps, Bonney forceps, DeBakey forceps, Russian forceps) [21] Clamps (locking forceps) Clamps stabilize or hold tissue and objects in place. [21] They can be used for traumatic or atraumatic purposes.
30 to 50% of stroke survivors develop post-stroke depression, which is characterized by lethargy, irritability, sleep disturbances, lowered self-esteem and withdrawal. [230] It is most common in those with a stroke affecting the anterior parts of the brain or the basal ganglia, particularly on the left side. [231]
Focal and diffuse brain injury are ways to classify brain injury: focal injury occurs in a specific location, while diffuse injury occurs over a more widespread area.It is common for both focal and diffuse damage to occur as a result of the same event; many traumatic brain injuries have aspects of both focal and diffuse injury. [1]
This is often a result of secondary injury, which can damage neurons that were unharmed in the primary injury. It occurs after a variety of brain injury including subarachnoid hemorrhage, stroke, and traumatic brain injury and involves metabolic cascades. [13] Secondary injury can result from complications of the injury. [1]
The relative risk of post-traumatic seizures (PTS) increases with the severity of traumatic brain injury (TBI). [128] A CT of the head years after a traumatic brain injury showing an empty space where the damage occurred marked by the arrow. Improvement of neurological function usually occurs for two or more years after the trauma.