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In addition to agenesis of the corpus callosum, similar conditions are hypogenesis (partial formation), dysgenesis (malformation), and hypoplasia (underdevelopment, including too thin). Other studies have also linked possible correlations between corpus callosum malformation and autism spectrum disorders. [32] [33]
Agenesis of the corpus callosum (ACC) is a rare birth defect in which there is a complete or partial absence of the corpus callosum. It occurs when the development of the corpus callosum, the band of white matter connecting the two hemispheres in the brain , in the embryo is disrupted.
Split-brain or callosal syndrome is a type of disconnection syndrome when the corpus callosum connecting the two hemispheres of the brain is severed to some degree. It is an association of symptoms produced by disruption of, or interference with, the connection between the hemispheres of the brain.
An example is commissural disconnect in adults which usually results from surgical intervention, tumor, or interruption of the blood supply to the corpus callosum or the immediately adjacent structures. Callosal disconnection syndrome is characterized by left ideomotor apraxia and left-hand agraphia and/or tactile anomia, and is relatively rare.
The corpus callosum is essential to the communication between the two hemispheres. [2] A recent study of individuals with agenesis of the corpus callosum suggests that the corpus callosum plays a vital role in problem solving strategies, verbal processing speed, and executive performance. Specifically, the absence of a fully developed corpus ...
Agenesis of the corpus callosum (ACC) is a rare, congenital brain defect that occurs when the corpus callosum does not develop normally. In majority of cases, ACC is caused by genetic factors. Genetic factors can include single gene mutations, multiple gene changes, and chromosomal abberation [ 3 ] There is a gap knowledge on how genetic ...
This condition is caused by X-linked recessive mutations in the L1CAM gene, located in the long arm of the X chromosome. [3] Mutations involved in the milder variants of L1 syndrome (including X-linked complicated corpus callosum dysgenesis) usually work by changing the L1 protein structure. [4]
Deep gray matter involvement commonly occurs in ADEM but is very rare in MS. Leptomeningeal involvement is not typical of either MS or ADEM: if 10 lesions are found in the brain of an MS patient, a lesion may be found in the corpus callosum. If a Susac patient has 10 lesions, more than half will be in the corpus callosum.