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Polyhydramnios is a medical condition describing an excess of amniotic fluid in the amniotic sac.It is seen in about 1% of pregnancies. [1] [2] [3] It is typically diagnosed when the amniotic fluid index (AFI) is greater than 24 cm. [4] [5] There are two clinical varieties of polyhydramnios: chronic polyhydramnios where excess amniotic fluid accumulates gradually, and acute polyhydramnios ...
Twin-to-twin transfusion syndrome (TTTS), also known as feto-fetal transfusion syndrome (FFTS), twin oligohydramnios-polyhydramnios sequence (TOPS) and stuck twin syndrome, is a complication of monochorionic multiple pregnancies (the most common form of identical twin pregnancy) in which there is disproportionate blood supply between the fetuses.
Complications from surgical repair include Peptic ulcer disease, Gastroesophageal reflux disease, Cholecystitis, Esophagitis, Megaduodenum, Blind loop syndrome, and anastomotic leak. [10] Late complications may occur in about 12 percent of patients with duodenal atresia, and the mortality rate for these complications is 6 percent.
When fetal kidneys begin to function around week 16, fetal urine also contributes to the fluid. [1] In earlier times, it was believed that the amniotic fluid was composed entirely of excreted fetal urine. The fluid is absorbed through the fetal tissue and skin. [2] After 22 to 25 week of pregnancy, keratinization of an embryo's skin occurs.
The fetal symptoms are related to fluid retention, including ascites and polyhydramnios. [3] Fetal hydrops suggests the presence of an important and probably fatal fetal pathology . It can be associated with parvovirus B19 infection and with twin-to-twin transfusion syndrome .
Gestational diabetes increases risk for further maternal and fetal complications such as development of pre-eclampsia, need for cesarean delivery, preterm delivery, polyhydramnios, macrosomia, shoulder dystocia, fetal hypoglycemia, hyperbilirubinemia, and admission into the neonatal intensive care unit.
The neonatal bowel obstruction is suspected based on polyhydramnios in utero, bilious vomiting, failure to pass meconium in the first day of life, and abdominal distension. [3] The presentations of NBO may vary. [ 4 ]
Complications in the baby may include premature birth, cord compression, and infection. [2] [1] Complications in the mother may include placental abruption and postpartum endometritis. [2] Risk factors include infection of the amniotic fluid, prior PROM, bleeding in the later parts of pregnancy, smoking, and a mother who is underweight. [2]