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Irving Glickman (January 17, 1914 – October 2, 1972) was an American clinical researcher described as "the father of periodontology" [1] and an author. He was one of the first to classify furcation defects and the role of occlusal trauma on periodontal disease.
Host modulatory therapy is an emerging treatment concept in the management of periodontitis that aims to reduce tissue destruction and stabilise or even regenerate the periodontium by modifying the host response.
In dentistry, numerous types of classification schemes have been developed to describe the teeth and gum tissue in a way that categorizes various defects. All of these classification schemes combine to provide the periodontal diagnosis of the aforementioned tissues in their various states of health and disease.
Plaque hypotheses are theories to explain the role of plaque bacteria in dental caries and in periodontal disease.They rely heavily on the postulates of Koch (formulated in 1884) and on the work of Louis Pasteur (1822–1895).
An internationally agreed classification formulated at the World Workshop in Clinical Periodontics in 1989 divided periodontal diseases into 5 groups: adult periodontitis, early-onset periodontitis, periodontitis associated with systemic disease, necrotizing ulcerative periodontitis and refractory periodontitis.
Gingival enlargement has a multitude of causes. The most common is chronic inflammatory gingival enlargement, when the gingivae are soft and discolored. This is caused by tissue edema and infective cellular infiltration caused by prolonged exposure to bacterial plaque, and is treated with conventional periodontal treatment, such as scaling and root planing.
The periodontium (from Greek περί (peri-) 'around' and -odont 'tooth') is the specialized tissues that both surround and support the teeth, maintaining them in the maxillary and mandibular bones.
In 1890, W.D. Miller, considered the father of oral microbiology, was the first to associate pulpal disease with the presence of bacteria. [11] This was confirmed by Kakehashi, who, in 1965, proved that bacteria were the cause of pulpal and periradicular disease in studies using animal models; pulpal exposures were initiated in both normal and germ-free rats, and while no pathologic changes ...