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[39] [37] The combination of an estrogen and 100 to 300 mg/day oral progesterone has been found to improve sleep outcomes. [39] [37] [47] Moreover, sleep was improved to a significantly better extent than estrogen plus medroxyprogesterone acetate. [39] This may be attributable to the sedative neurosteroid effects of progesterone. [39]
The endometrial transformation dosage of oral micronized progesterone in women has been listed as 200 to 300 mg/day or 4,200 mg total per cycle. [ 116 ] [ 1 ] However, a clinical study found that 300 mg/day oral micronized progesterone was insufficient for full endometrial transformation. [ 117 ]
For comparison to MPA, the dosage of progesterone required to inhibit ovulation is 300 mg/day, whereas that of the 19-nortestosterone derivatives norethisterone and norethisterone acetate is only 0.4 to 0.5 mg/day.
[83] [68] Even 600 mg/day oral progesterone, which is a very high dosage, fails to produce full luteal-phase endometrial changes, [71] although doses of 300 to 600 mg/day oral progesterone have reportedly been used for luteal support in assisted reproduction. [68]
Ethinylestradiol and norethisterone acetate (FemHRT) – 25 μg / 0.5 mg; Estradiol/progesterone (TX-001HR), a combination of estradiol and progesterone in oil-filled capsules, is currently pending approval. [45] Estradiol and norgestimate (Prefest; 1 mg / 90 μg) was previously available in the U.S. but was discontinued.
In 1953, at Pincus' suggestion, Rock induced a three-month anovulatory "pseudopregnancy" state in twenty-seven of his infertility patients with an oral 300 mg/day progesterone-only regimen for 20 days from cycle days 5–24 followed by pill-free days to produce withdrawal bleeding. [191]