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Phosgene was first deployed as a chemical weapon by the French in 1915 in World War I. [24] It was also used in a mixture with an equal volume of chlorine, with the chlorine helping to spread the denser phosgene. [25] [26] Phosgene was more potent than chlorine, though some symptoms took 24 hours or more to manifest.
A possible use of phosphenes as part of a brain to brain communication system has been reported. The system called BrainNet, produces phosphenes using transcranial magnetic stimulation (TMS). The goal of the research is to connect thoughts brain to brain using a system where signals are detected using electroencephalography ( EEG ) and ...
Some elements of the cholinergic crisis can be reversed with antimuscarinic drugs like atropine or diphenhydramine, but the most dangerous effect - respiratory depression, cannot. [ 6 ] The neuromuscular junction, where the brain communicates with muscles (like the diaphragm , the main breathing muscle), works by acetylcholine activating ...
Standard treatment for nerve agent poisoning is a combination of an anticholinergic to manage the symptoms, and an oxime as an antidote. [12] Anticholinergics treat the symptoms by reducing the effects of acetylcholine, while oximes displaces phosphate molecules from the active site of the cholinesterase enzymes, allowing the breakdown of ...
All symptoms listed above are consistent with mold mycotoxin accumulation. [ 7 ] The term neurotoxicity implies the involvement of a neurotoxin; however, the term neurotoxic may be used more loosely to describe states that are known to cause physical brain damage , but where no specific neurotoxin has been identified.
The CDC says symptoms of carbon monoxide poisoning are "variable and nonspecific," but include "headache, dizziness, weakness, nausea, vomiting, chest pain, and altered mental status."
Toxic encephalopathy is a neurologic disorder caused by exposure to neurotoxic organic solvents such as toluene, following exposure to heavy metals such as manganese, as a side effect of melarsoprol treatment for African trypanosomiasis, adverse effects to prescription drugs, or exposure to extreme concentrations of any natural toxin such as cyanotoxins found in shellfish or freshwater ...
Initial symptoms following exposure to sarin are a runny nose, tightness in the chest, and constriction of the pupils (miotic action). Soon after, the person will have difficulty breathing and experience nausea and drooling. This progresses to losing control of bodily functions, which may cause the person to vomit, defecate, and urinate.