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2. Acetylcarnitine - Wikipedia

   en.wikipedia.org/wiki/Acetylcarnitine

   Acetyl-L-carnitine, ALCAR or ALC, is an acetylated form of L-carnitine.It is naturally produced by the human body, and it is available as a dietary supplement. Acetylcarnitine is broken down in the blood by plasma esterases to carnitine which is used by the body to transport fatty acids into the mitochondria for breakdown and ener

3. Carnitine-acylcarnitine translocase deficiency - Wikipedia

   en.wikipedia.org/wiki/Carnitine-acylcarnitine...

   Carnitine-acylcarnitine translocase deficiency is a rare, autosomal recessive metabolic disorder that prevents the body from converting long-chain fatty acids into energy, particularly during periods without food. [1] Carnitine, a natural substance

4. Carnitine - Wikipedia

   en.wikipedia.org/wiki/Carnitine

   L-Carnitine, acetyl-l-carnitine, and propionyl-l-carnitine are available in dietary supplement pills or powders, with a daily amount of 0.5 to 1 g considered to be safe. [1] [3] It is also a drug approved by the Food and Drug Administration to treat primary and certain secondary carnitine-deficiency syndromes secondary to inherited diseases. [1 ...

5. Carnitine-acylcarnitine translocase - Wikipedia

   en.wikipedia.org/wiki/Carnitine-acylcarnitine...

   This disorder disrupts the carnitine shuttle system from moving fatty acids across the mitochondrial membrane, leading to a decrease in fatty acid catabolism. The result is an accumulation of fatty acid within muscles and liver, decreased tolerance to long term exercise, inability to fast for more than a few hours, muscle weakness and wasting ...

6. Medium-chain acyl-coenzyme A dehydrogenase deficiency

   en.wikipedia.org/wiki/Medium-chain_acyl-coenzyme...

   Medium-chain acyl-CoA dehydrogenase deficiency (MCAD deficiency or MCADD) is a disorder of fatty acid oxidation that impairs the body's ability to break down medium-chain fatty acids into acetyl-CoA. The disorder is characterized by hypoglycemia and sudden death without timely intervention, most often brought on by periods of fasting or vomiting.

7. Ketogenesis - Wikipedia

   en.wikipedia.org/wiki/Ketogenesis

   Without insulin to help extract glucose from the blood, tissues the levels of malonyl-CoA are reduced, and it becomes easier for fatty acids to be transported into mitochondria, causing the accumulation of excess acetyl-CoA. The accumulation of acetyl-CoA in turn produces excess ketone bodies through ketogenesis. [11]