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The T4 virus initiates an Escherichia coli infection by binding OmpC porin proteins and lipopolysaccharide (LPS) on the surface of E. coli cells with its long tail fibers (LTF). [16] [17] A recognition signal is sent through the LTFs to the baseplate. This unravels the short tail fibers (STF) that bind irreversibly to the E. coli cell surface.
The virus attaches to the host cell using its terminal fibers, and uses viral exolysin to degrade the cell wall enough to eject the viral DNA into the host cytoplasm via contraction of its tail sheath. DNA-templated transcription is the method of transcription. The virus exits the host cell by lysis, and holin/endolysin/spanin proteins. Once ...
Escherichia virus T4; Enterobacteria phage T6; Escherichia virus T3; Escherichia virus T5; T. T4 rII system; T7 DNA helicase; T7 DNA polymerase; T7 phage; T7 RNA ...
Researchers in the United States and United Kingdom have shown how the bacteria responsible for many urinary tract infections have adapted to prevent being flushed out when you pee.
The first phages that were studied in detail included seven that commonly infect E. coli. They were named Type 1 (T1), Type 2 (T2), etc., for easy reference; however, due to structural similarities between the T2, T4, and T6 bacteriophages, these are now commonly referred to as T-Even phages.
Schematic drawings of a phage virion (species Escherichia virus T4, cross sections and side view) Viruses in Tevenvirinae are non-enveloped, with head-tail geometries. These viruses are about 70 nm wide and 140 nm long. Genomes are linear, around 170-245kb in length. The genome codes for 300 to 415 proteins. [3]
Enterobacteria phage T6 is a bacteriophage strain that infects Escherichia coli bacteria. It was one bacteriophage that was used as a model system in the 1950s in exploring the methods viruses replicate, along with the other T-even bacteriophages (which build up virus species Escherichia virus T4, a member of genus T4virus according to ICTV nomenclature): [1] Enterobacteria phage T2 ...
Organisms commonly producing this infection are Streptococcus pyogenes; staphylococci (inhabitants of the skin and of pimples, carbuncles, and many other pustular eruptions); the anaerobic streptococci, which flourish in devitalized tissues such as may be present after long and injurious labour and unskilled instrumental delivery; Escherichia ...