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Pseudomonas syringae is a rod-shaped, Gram-negative bacterium with polar flagella.As a plant pathogen, it can infect a wide range of species, and exists as over 50 different pathovars, [2] all of which are available to researchers from international culture collections such as the NCPPB, ICMP, and others.
"Pseudomonas tomato" is a Gram-negative plant pathogenic bacterium that infects a variety of plants. It was once considered a pathovar of Pseudomonas syringae , but following DNA-relatedness studies, it was recognized as a separate species and several other former P. syringae pathovars were incorporated into it. [ 1 ]
In 2000, the complete genome sequence of a Pseudomonas species was determined; more recently, the sequence of other strains has been determined, including P. aeruginosa strains PAO1 (2000), P. putida KT2440 (2002), P. protegens Pf-5 (2005), P. syringae pathovar tomato DC3000 (2003), P. syringae pathovar syringae B728a (2005), P. syringae ...
Several pathovars of Pseudomonas syringae have been sequenced, including pathovar tomato DC3000 (2003), pathovar syringae B728a (2005), and pathovar phaseolica 1448A (2005). [ 3 ] Distinguishing characteristics
Pseudomonas syringae pv. tomato: Bacterial spot: Xanthomonas campestris pv. vesicatoria: Bacterial stem rot and fruit rot Erwinia carotovora subsp. carotovora: Bacterial wilt: Ralstonia solanacearum: Pith necrosis Pseudomonas corrugata: Syringae leaf spot Pseudomonas syringae pv. syringae: Aster yellows Ca. Phytoplasma asteris Tomato big bud Ca.
Pseudomonas syringae. P. syringae pv. syringae. Crown Gall ... Sinaloa tomato leaf curl genus Begomovirus, Sinaloa tomato leaf curl virus (TCLV) Tobacco etch
Tomato Bacterial Spot is caused by Xanthomonas axonopodis pv. vesicatoria. Tomato Bacterial Speck is produced by Pseudomonas syringae pv. tomato. Both are economically significant in fresh-market tomato here. [4]
AvrPto is an ancient effector that is conserved in many P. syringae strains, whereas Pto R gene is only found in a few wild tomato species. [18] This suggests recent evolution of the Pto R gene and the pressure to evolve to target AvrPto, turning a virulence effector to an avirulence effector.