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The anterior spinothalamic tract (Latin: tractus spinothalamicus anterior) or ventral spinothalamic fasciculus situated in the marginal part of the anterior funiculus and intermingled more or less with the vestibulospinal tract, is derived from cells in the posterior column or intermediate gray matter of the opposite side.
The dorsal column–medial lemniscus pathway (DCML) (also known as the posterior column-medial lemniscus pathway (PCML) is the major sensory pathway of the central nervous system that conveys sensations of fine touch, vibration, two-point discrimination, and proprioception (body position) from the skin and joints.
These axons enter the spinal column and penetrate the grey matter of the dorsal horn, where they synapse on second-order neurons in either the substantia gelatinosa of Rolando or the nucleus proprius. Those neurons project their axon to the anterolateral quadrant of the contralateral half of the spinal cord, where they give the spinothalamic tract.
This tract was historically considered a cephalic division of the medial lemniscus due to the close proximity of the two ascending tracts. [2] Like the medial lemniscus in the dorsal column-medial lemniscus pathway (DCML), that carries mechanosensory information from part of the head and the rest of the body, the trigeminal lemniscus carries ...
It is part of the posterior column-medial lemniscus pathway, which transmits touch, vibration sense, as well as the pathway for proprioception. [citation needed] The medial lemniscus carries axons from most of the body and terminates by synapsing with third-order neurons in the ventral posterolateral nucleus of the thalamus.
This means that a lesion of the dorsal columns will cause loss of touch and proprioception below the lesion and on the same side as it, while a lesion of the spinothalamic tracts will cause loss of pain and temperature below the lesion and on the opposite side to it. [1]
Subacute combined degeneration of spinal cord, also known as myelosis funiculus, or funicular myelosis, [1] also Lichtheim's disease, [2] [3] and Putnam-Dana syndrome, [4] refers to degeneration of the posterior and lateral columns of the spinal cord as a result of vitamin B 12 deficiency (most common).
The ventral posterolateral nucleus (VPL) is one of the subdivisions of the ventral posterior nucleus in the ventral nuclear group of the thalamus. [1] It relays sensory information from the second-order neurons of the neospinothalamic tract and medial lemniscus (of the dorsal column-medial lemniscus pathway) which synapse with the third-order neurons in the nucleus.