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Japanese encephalitis (JE) is an infection of the brain caused by the Japanese encephalitis virus (JEV). [3] While most infections result in little or no symptoms, occasional inflammation of the brain occurs. [3] In these cases, symptoms may include headache, vomiting, fever, confusion and seizures. [1] This occurs about 5 to 15 days after ...
Japanese encephalitis vaccines first became available in the 1930s. [3] One of them was an inactivated mouse brain-derived vaccine (the Nakayama and/or Beijing-1 strain), made by BIKEN and marketed by Sanofi Pasteur as JE-VAX, until production ceased in 2005. The other was an inactivated vaccine cultivated on primary hamster kidney cells (the ...
Encephalitis with meningitis is known as meningoencephalitis, while encephalitis with involvement of the spinal cord is known as encephalomyelitis. [ 2 ] The word is from Ancient Greek ἐγκέφαλος , enképhalos 'brain', [ 37 ] composed of ἐν , en , 'in' and κεφαλή , kephalé , 'head', and the medical suffix -itis 'inflammation'.
For some forms of viral encephalitis, such as Eastern equine encephalitis and Japanese encephalitis, there may be a significant amount of necrosis of nerve cells. Following encephalitis caused by arboviruses, calcification may occur in the CNS, especially among children. Herpes simplex encephalitis tends to produce necrotic lesions in the CNS. [1]
Dengue fever, Japanese encephalitis, other hemorrhagic fevers: Regional arbovirus infections: California serogroup virus, Eastern equine encephalitis virus, Powassan virus, St. Louis encephalitis virus, Western equine encephalitis virus: Varicella voster infection- chickenpox, shingles, and unspecified Chickenpox: Chickenpox (regional)
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Life cycle of Japanese encephalitis virus (JEV) Viral replication is cytoplasmic. Entry into the host cell is achieved by attachment of the viral envelope protein E to host receptors, which mediates clathrin-mediated endocytosis. Replication follows the positive-stranded RNA virus replication model.