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Hemispatial neglect is a neuropsychological condition in which, after damage to one hemisphere of the brain (e.g. after a stroke), a deficit in attention and awareness towards the side of space opposite brain damage (contralesional space) is observed.
Weber's syndrome, also known as midbrain stroke syndrome or superior alternating hemiplegia, is a form of stroke that affects the medial portion of the midbrain. It involves oculomotor fascicles in the interpeduncular cisterns and cerebral peduncle so it characterizes the presence of an ipsilateral lower motor neuron type oculomotor nerve palsy and contralateral hemiparesis or hemiplegia.
Hemiparesis, also called unilateral paresis, is the weakness of one entire side of the body (hemi-means "half"). Hemiplegia , in its most severe form, is the complete paralysis of one entire side of the body.
In medicine, paresis (/ p ə ˈ r iː s ɪ s, ˈ p æ r ə s ɪ s /), compound word from Greek Ancient Greek: πάρεσις, (πᾰρᾰ- “beside” + ἵημι “let go, release”), is a condition typified by a weakness of voluntary movement, or by partial loss of voluntary movement or by impaired movement.
Superior alternating hemiplegia (also known as Weber syndrome) has a few distinct symptoms: contralateral hemiparesis of limb and facial muscle accompanied by weakness in one or more muscles that control eye movement on the same side. [2] Another symptom that appears is the loss of eye movement due to damage to the oculomotor nerve fibers.
Claude's syndrome is a form of brainstem stroke syndrome characterized by the presence of an ipsilateral oculomotor nerve palsy, contralateral hemiparesis, contralateral ataxia, and contralateral hemiplegia of the lower face, tongue, and shoulder.
Contralateral spastic hemiparesis: Medial lemniscus: Contralateral PCML (aka DCML) pathway loss (tactile, vibration, and stereognosis) Abducens nerve: Strabismus (ipsilateral lateral rectus muscle paralysis - the affected eye looks down and towards the nose). Abducens nerve lesion localizes the lesion to inferior pons.
Contralateral loss of sensory modalities in the body (damage to spinothalamic tract and medial lemniscus) Contralateral hemiparesis of face and body (damage to corticospinal tract) may occur with ventral extension of lesion; Horizontal gaze palsy may occur (as in lower dorsal pontine syndrome)