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The bladder also contains β 3 adrenergic receptors, and pharmacological agonists of this receptor are used to treat overactive bladder. The mucosa of the urinary bladder may herniate through the detrusor muscle. [6] This is most often an acquired condition due to high pressure in the urinary bladder, damage, or existing connective tissue ...
Neurogenic bladder dysfunction, often called by the shortened term neurogenic bladder, refers to urinary bladder problems due to disease or injury of the central nervous system or peripheral nerves involved in the control of urination. [1] [2] There are multiple types of neurogenic bladder depending on the underlying cause and the symptoms.
It is located at the junction of the urethra with the urinary bladder and is continuous with the detrusor muscle, [1] [2] but anatomically and functionally fully independent from it. [3] It is composed of smooth muscle , so it is under the control of the autonomic nervous system , specifically the sympathetic nervous system .
Percutaneous tibial nerve stimulation (PTNS), also referred to as posterior tibial nerve stimulation, is the least invasive form of neuromodulation used to treat overactive bladder (OAB) and the associated symptoms of urinary urgency, urinary frequency and urge incontinence.
Bladder location and associated structures in the male. In males, the prostate gland lies outside the opening for the urethra. The middle lobe of the prostate causes an elevation in the mucous membrane behind the internal urethral orifice called the uvula of urinary bladder. The uvula can enlarge when the prostate becomes enlarged.
The urethra (pl.: urethras or urethrae) is the tube that connects the urinary bladder to the urinary meatus, [1] [2] through which placental mammals urinate and ejaculate. [3] In non-mammalian vertebrates, the urethra also transports semen but is separate from the urinary tract.
[1] [2] Symptoms depend on the site of nerve damage and can include motor changes such as weakness; sensory symptoms such as numbness, tingling, or pain; or autonomic changes such as urinary symptoms. These changes are thought to result from a microvascular injury involving small blood vessels that supply nerves (vasa nervorum).
The arteries that supply the ureters end in a network of vessels within the adventitia of the ureters. [1] There are many connections (anastamoses) between the arteries of the ureter, [2] particularly in the adventitia, [5] which means damage to a single vessel does not compromise the blood supply of the ureter.