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Left ventricular hypertrophy with secondary repolarization abnormalities as seen on ECG Histopathology of (a) normal myocardium and (b) myocardial hypertrophy. Scale bar indicates 50 μm. Gross pathology of left ventricular hypertrophy. Left ventricle is at right in image, serially sectioned from apex to near base.
Left axis deviation symptoms depend on the underlying cause. [5] For example, if left ventricular hypertrophy is the cause of LAD, symptoms can include shortness of breath, fatigue, chest pain (especially with exercise), palpitations, dizziness, or fainting. [6]
In electrocardiography, a strain pattern is a well-recognized marker for the presence of anatomic left ventricular hypertrophy (LVH) in the form of ST depression and T wave inversion on a resting ECG. [1] It is an abnormality of repolarization and it has been associated with an adverse prognosis in a variety heart disease patients.
Ventricular hypertrophy (VH) is thickening of the walls of a ventricle (lower chamber) of the heart. [ 1 ] [ better source needed ] Although left ventricular hypertrophy (LVH) is more common, right ventricular hypertrophy (RVH), as well as concurrent hypertrophy of both ventricles can also occur.
Regarding differential diagnosis, left ventricular hypertrophy is usually indistinguishable from athlete's heart and at ECG, but can usually be discounted in the young and fit. [ 15 ] [ 16 ] It is important to distinguish between athlete's heart and hypertrophic cardiomyopathy (HCM), a serious cardiovascular disease characterized by thickening ...
Left anterior fascicular block (LAFB) is an abnormal condition of the left ventricle of the heart, [1] [2] related to, but distinguished from, left bundle branch block (LBBB). It is caused by only the left anterior fascicle – one half of the left bundle branch being defective. It is manifested on the ECG by left axis deviation.
Left Ventricular Hypertrophy. Left ventricular hypertrophy (LVH) is an increase in the thickness and mass of the myocardium. This could be a normal reversible response to cardiovascular conditioning (athletic heart) or an abnormal irreversible response to chronically increased volume load (preload) or increased pressure load (afterload).
The diagnosis of left ventricular outflow tract obstruction is usually made by echocardiographic assessment and is defined as a peak left ventricular outflow tract gradient of ≥ 30 mmHg. [ 37 ] Another, non-obstructive variant of HCM is apical hypertrophic cardiomyopathy ( AHCM or ApHCM ), [ 39 ] also called Yamaguchi syndrome .
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