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2. Beta-adrenergic agonist - Wikipedia

   en.wikipedia.org/wiki/Beta-adrenergic_agonist

   Epinephrine (adrenaline). Activation of β 1 receptors induces positive inotropic, chronotropic output of the cardiac muscle, leading to increased heart rate and blood pressure, secretion of ghrelin from the stomach, and renin release from the kidneys.

3. Cardiotonic agent - Wikipedia

   en.wikipedia.org/wiki/Cardiotonic_agent

   Cardiotonic agents are typically employed as short-term and non-routine therapies for heart failure patients. [71] They are specifically reserved for those with contractile dysfunction in the left ventricles (lower chambers of the heart), low cardiac output, and low blood pressure, placing them at risk of inadequate organ perfusion. [72]

4. Epinephrine (medication) - Wikipedia

   en.wikipedia.org/wiki/Epinephrine_(medication)

   Epinephrine, also known as adrenaline, is a medication and hormone. [10] [11] As a medication, it is used to treat several conditions, including anaphylaxis, cardiac arrest, asthma, and superficial bleeding. [8] Inhaled epinephrine may be used to improve the symptoms of croup. [12] It may also be used for asthma when other treatments are not ...

5. What is a beta blocker? Why they are vital to those with ...

   www.aol.com/beta-blocker-why-vital-those...

   They're prescribed to people with some irregular heart rhythms, heat attacks, heart failure, chest pain and other cardiovascular problems. Churchwell added beta blockers are one of the most ...

6. Beta1-adrenergic agonist - Wikipedia

   en.wikipedia.org/wiki/Beta1-adrenergic_agonist

   As a result, they act more selectively upon the heart. β-Adrenoceptors typically bind to norepinephrine release by sympathetic adrenergic nerves and to circulating epinephrine. The effect of β-adrenoceptors is cardiac stimulation, such as increased heart rate, heart contractility, heart conduction velocity, and heart relaxation. [1]

7. Beta blocker - Wikipedia

   en.wikipedia.org/wiki/Beta_blocker

   Heart failure characteristically involves increased catecholamine activity on the heart, which is responsible for several deleterious effects, including increased oxygen demand, propagation of inflammatory mediators, and abnormal cardiac tissue remodeling, all of which decrease the efficiency of cardiac contraction and contribute to the low ...