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Ventilation–perfusion coupling is the relationship between ventilation and perfusion processes, which take place in the respiratory system and the cardiovascular system. [1] Ventilation is the movement of gas during breathing, and perfusion is the process of pulmonary blood circulation , which delivers oxygen to body tissues. [ 2 ]
The diffusing capacity does not directly measure the primary cause of hypoxemia, or low blood oxygen, namely mismatch of ventilation to perfusion: [2] Not all pulmonary arterial blood goes to areas of the lung where gas exchange can occur (the anatomic or physiologic shunts), and this poorly oxygenated blood rejoins the well oxygenated blood ...
Mucociliary clearance (MCC), mucociliary transport, or the mucociliary escalator describes the self-clearing mechanism of the airways in the respiratory system. [1] It is one of the two protective processes for the lungs in removing inhaled particles including pathogens before they can reach the delicate tissue of the lungs.
It is in effect an external extension of the physiological dead space. [10] It can be reduced by: Using separate intake and exhaust passages with one-way valves placed in each passage, usually between each passage and the mouthpiece. This limits the dead space to between the non return valves and the user's mouth or nose.
Ventilation Scan is abnormal but perfusion scan is normal indicating abnormal airway suggesting COPD or asthma. Ventilation Scan is normal but perfusion is abnormal indicating any obstruction to the blood flow (perfusion), may be because of the pulmonary embolism obstructing the flow. Both scans are abnormal. It may be found in pneumonia or COPD.
Any process that increases the CC by increasing the closing volume (CV) can increase an individual's risk of hypoxemia, as the small airways may collapse during exhalation, leading to air trapping and atelectasis. A mnemonic for factors increasing closing capacity is ACLS-S: Age, Chronic bronchitis, LV failure, Smoking, Surgery.
Short term hypocapnia does not usually have any adverse effects. [2] It is sometimes used as lifesaving treatment for conditions such as neonatal pulmonary-artery hypertension and for people with severe intracranial hypertension. If the state of hypocapnia persists or is prolonged, adverse outcomes may occur. [2]
Factors that can increase the D LCO include polycythaemia, asthma (can also have normal D LCO) and increased pulmonary blood volume as occurs in exercise.Other factors are left to right intracardiac shunting, mild left heart failure (increased blood volume) and alveolar hemorrhage (increased blood available for which CO does not have to cross a barrier to enter).
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