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The Journal of Asthma and Allergy is a peer-reviewed medical journal focusing on asthma and pulmonary physiology. The journal was established in 2008 and is published by Dove Medical Press . External links
The mechanisms behind allergic asthma—i.e., asthma resulting from an immune response to inhaled allergens—are the best understood of the causal factors. In both people with asthma and people who are free of the disease, inhaled allergens that find their way to the inner airways are ingested by a type of cell known as antigen-presenting ...
Asthma triggers are factors or stimuli that provoke the exacerbation of asthma symptoms or increase the degree of airflow disruption, which can lead to an asthma attack. [1] An asthma attack is characterized by an obstruction of the airway , hypersecretion of mucus and bronchoconstriction due to the contraction of smooth muscles around the ...
The Journal of Asthma is a peer-reviewed medical journal that covers asthma and related conditions. The editor-in-chief is Fulvio Braido ( University of Genoa ). [ 1 ]
Asthma phenotyping and endotyping is a novel approach to asthma classification inspired by precision medicine.It seeks to separate the clinical presentations or clusters of signs and symptoms of asthma, known as asthma phenotypes, from their underlying etiologies or causes, known as asthma endotypes.
Allergy, Asthma & Immunology Research is a bimonthly peer-reviewed open access medical journal covering immunology. It was established in 2009 and is published by the Korean Academy of Asthma, Allergy and Clinical Immunology and the Korean Academy of Pediatric Allergy and Respiratory Disease; it is an official journal of both societies. [ 1 ]
Asthma phenotyping and endotyping has emerged as a novel approach to asthma classification inspired by precision medicine which separates the clinical presentations of asthma, or asthma phenotypes, from their underlying causes, or asthma endotypes. The best-supported endotypic distinction is the type 2-high/type 2-low distinction.
Airway remodelling is a multifaceted process involving multiple airway tissues. These include goblet cell hyperplasia, leading to increased mucus production, and airway smooth muscle hypertrophy (or smooth muscle cell hyperplasia), leading to the release of pro-inflammatory and pro-fibrotic messengers contributing to subepithelial fibrosis.