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(H35.0) Hypertensive retinopathy — burst blood vessels, due to long-term high blood pressure (H35.0/E10-E14) Diabetic retinopathy — damage to the retina caused by complications of diabetes mellitus, which could eventually lead to blindness (H35.0-H35.2) Retinopathy — general term referring to non-inflammatory damage to the retina
Diabetic angiopathy is a form of angiopathy associated with diabetic complications. [1] While not exclusive, the two most common forms are diabetic retinopathy and diabetic nephropathy, whose pathophysiologies are largely identical. Other forms of diabetic angiopathy include diabetic neuropathy and diabetic cardiomyopathy. [2] [3]
Around 35% of people with diabetes have some kind of diabetic retinopathy; around 10% experience some degree of vision loss. [58] Diabetic retinopathy is particularly common in those with type 1 diabetes – affecting 25% of people five years from diagnosis, 60% 10 years from diagnosis, and 80% 15 years from diagnosis. [59]
ICD-10 is the 10th revision of the International Classification of Diseases (ICD), a medical classification list by the World Health Organization (WHO). It contains codes for diseases, signs and symptoms, abnormal findings, complaints, social circumstances, and external causes of injury or diseases. [1]
The vast majority of AIONs are nonarteritic AIONs (NAION). The most common acute optic neuropathy in patients over 50 years of age, NAION has an annual incidence of 2.3-10.2/100,000. NAION presents as a painless loss of vision, often when awakening, that occurs over hours to days.
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Diabetes is the most common cause of retinopathy in the U.S. as of 2008. [4] Diabetic retinopathy is the leading cause of blindness in working-aged people. [ 5 ] It accounts for about 5% of blindness worldwide and is designated a priority eye disease by the World Health Organization.
Diabetic papillopathy is a self-limiting disease that may affect both type 1 and type 2 diabetic patients. Unilateral or bilateral optic disc edema may occur. The exact etiology, pathogenesis and mechanism of the disc edema is unknown.