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Syringomyelia is a generic term referring to a disorder in which a cyst or cavity forms within the spinal cord. Often, syringomyelia is used as a generic term before an etiology is determined. [ 3 ] This cyst, called a syrinx , can expand and elongate over time, destroying the spinal cord.
Syringomyelia; Hydromyelia. In hydromyelia, a dilation of the central canal of the spinal cord is caused by an increase of cerebrospinal fluid. [7] Syringohydromyelia (i.e., both Syringomyelia and Hydromyelia) [7] Tethered cord; In some cases, the terminal ventricle may cause clinical symptoms due to its expansion.
Tethering may also develop after spinal cord injury. Scar tissue can block the flow of fluids around the spinal cord. Fluid pressure may cause cysts to form in the spinal cord, a condition called syringomyelia. This can lead to additional loss of movement or feeling, or the onset of pain or autonomic nervous system symptoms. [6]
Diastematomyelia is a "dysraphic state" of unknown embryonic origin, but is probably initiated by an accessory neurenteric canal (an additional embryonic spinal canal.).) This condition may be an isolated phenomenon or may be associated with other segmental anomalies of the vertebral bodies such as spina bifida, kyphoscoliosis, butterfly vertebra, hemivertebra and block vertebrae which are ...
Cerebrospinal fluid fills the syrinx. Pressure differences along the spine cause the fluid to move within the cyst. Physicians believe that it is this continual movement of fluid that results in cyst growth and further damage to the spinal cord. In the case of syringomyelia, the syrinx can expand and elongate over time, destroying the spinal ...
A fluid-filled lesion forms, known as a syrinx. [1] This can vary in size significantly between patients. [1] Nerve fibres may be compressed where they cross the midline, or in other parts of the spinal cord. [1] Cranial nerves may be affected. [3] Syringobulbia may be associated with syringomyelia, a syrinx limited to the spinal cord. [4]
Type 1 is the most common form among the 5 types of HSAN. Its historical names include mal perforant du pied, ulcero-mutilating neuropathy, hereditary perforating ulcers, familial trophoneurosis, familial syringomyelia, hereditary sensory radicular neuropathy, among others. [3]
The location of cord lesions affects presentation—for instance, a central lesion (such as that of syringomyelia) will knock out second order neurons of the spinothalamic tract as they cross the centre of the cord, and will cause loss of pain and temperature without loss of fine touch or proprioception.