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The patient was a 48-year-old man with hypothyroidism, multiple episodes of encephalopathy, stroke-like symptoms, and Hashimoto's thyroiditis confirmed by elevated antithyroid antibodies. Alternative names
Myxedema psychosis is a relatively uncommon consequence of hypothyroidism, such as in Hashimoto's thyroiditis or in patients who have had the thyroid surgically removed and are not taking thyroxine. A chronically under-active thyroid can lead to slowly progressive dementia , delirium , and in extreme cases to hallucinations , coma , or ...
Hashimoto’s disease is an autoimmune condition in which your immune system mistakenly attacks this gland, reducing thyroid hormone production and symptoms that affect your whole body.
[8] [9] Hashimoto's thyroiditis is the most common cause of hypothyroidism in countries with sufficient dietary iodine. [3] Less common causes include previous treatment with radioactive iodine , injury to the hypothalamus or the anterior pituitary gland, certain medications, a lack of a functioning thyroid at birth , or previous thyroid surgery .
Replacement occurred faster in the United Kingdom than in North America, but by the 1980s more patients were being prescribed synthetic T4 (levothyroxine) or synthetic T4/T3 combinations than desiccated thyroid extract. [citation needed] Several reasons have been identified as to why prescriptions changed from desiccated thyroid treatment.
Additionally, patients may present with a variety of symptoms such as palpitations and arrhythmias (notably atrial fibrillation), shortness of breath , loss of libido, nausea, vomiting, and diarrhea. Long term untreated hyperthyroidism can lead to osteoporosis. In the elderly, these classical symptoms may not be present. [citation needed]
Hashimoto's thyroiditis, also known as chronic lymphocytic thyroiditis, Hashimoto's disease and autoimmune thyroiditis, is an autoimmune disease in which the thyroid gland is gradually destroyed. [7] [1] Early on, symptoms may not be noticed. [3] Over time, the thyroid may enlarge, forming a painless goiter. [3]
[22] [23] 3,5-T2 levels were also observed to correlate with concentrations of rT3 (reverse T3) [22] in patients with euthyroid sick syndrome. NTIS is a component of a complex endocrine adaptation process, so affected patients might also have hyperprolactinemia and elevated levels of corticosteroids (especially cortisol) and growth hormone.
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