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Traumatic brain injury (TBI, physical trauma to the brain) can cause a variety of complications, health effects that are not TBI themselves but that result from it. The risk of complications increases with the severity of the trauma; [1] however even mild traumatic brain injury can result in disabilities that interfere with social interactions, employment, and everyday living. [2]
Traumatic brain injury may cause a range of serious coincidental complications that include cardiac arrhythmias [117] and neurogenic pulmonary edema. [118] These conditions must be adequately treated and stabilised as part of the core care. Surgery can be performed on mass lesions or to eliminate objects that have penetrated the brain.
Although 20-33% of healthy elderly adults have these deposits, they are increased in elderly with diagnosed Alzheimer's disease and dementia. [35] Amyloid plaques associated with Alzheimer's disease that increase in number with age. Additionally, traumatic brain injury, TBI, is increasingly being linked as a factor in early-onset Alzheimer's ...
Even a mild incident can have long-term effects or cause symptoms to appear years later. [5] Studies show there is a correlation between brain lesion and language, speech, and category-specific disorders. Wernicke's aphasia is associated with anomia, unknowingly making up words , and problems with comprehension.
A concussion, also known as a mild traumatic brain injury (mTBI), is a head injury that temporarily affects brain functioning. [8] Symptoms may include headache, dizziness, difficulty with thinking and concentration, sleep disturbances, mood changes, a brief period of memory loss, brief loss of consciousness; problems with balance; nausea; blurred vision; and mood changes.
Traumatic brain injury may cause damage to the hypothalamus or the pituitary gland, and deficiencies of pituitary hormones (hypopituitarism) can cause similar symptoms to post-concussion syndrome; in these cases, symptoms can be treated by replacing any hormones that are deficient. [medical citation needed]
PTS are more likely to occur in more severe injuries, and certain types of injuries increase the risk further. The risk that a person will develop PTS becomes progressively lower as time passes after the injury. However, TBI survivors may still be at risk over 15 years after the injury. [5] Children and older adults are at a higher risk for PTS.
This is often a result of secondary injury, which can damage neurons that were unharmed in the primary injury. It occurs after a variety of brain injury including subarachnoid hemorrhage, stroke, and traumatic brain injury and involves metabolic cascades. [13] Secondary injury can result from complications of the injury. [1]
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