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Diastolic heart murmurs are heart murmurs heard during diastole, [1] [2] [3] i.e. they start at or after S2 and end before or at S1. Many involve stenosis of the atrioventricular valves or regurgitation of the semilunar valves .
Heart murmurs are unique heart sounds produced when blood flows across a heart valve or blood vessel. [1] This occurs when turbulent blood flow creates a sound loud enough to hear with a stethoscope. [2] The sound differs from normal heart sounds by their characteristics. For example, heart murmurs may have a distinct pitch, duration and timing.
The first heart sound, or S 1, forms the "lub" of "lub-dub" and is composed of components M 1 (mitral valve closure) and T 1 (tricuspid valve closure). Normally M 1 precedes T 1 slightly. It is caused by the closure of the atrioventricular valves , i.e. tricuspid and mitral (bicuspid), at the beginning of ventricular contraction, or systole .
The valves in your heart act as doors between the chambers, or rooms, of the heart. In the case of a murmur, a valve may be tight or leaky. When heart valves are narrow, this is called stenosis.
Dr. Davis emphasizes that valve damage is not only caused by poor dietary habits, explaining, “Diet is often talked about in connection to heart disease, but valve damage has different causes.
Mitral stenosis is a valvular heart disease characterized by the narrowing of the opening of the mitral valve of the heart. [1] It is almost always caused by rheumatic valvular heart disease . Normally, the mitral valve is about 5 cm 2 during diastole.
The mitral valve in cases of mitral stenosis may open with an opening snap [1] [2] on the beginning of diastole. Patients with mitral valve prolapse may have a mid-systolic click along with a murmur, referred to as apical late systolic murmur. [3] Early systolic clicks may also be present in some patients. [4]
The fourth heart sound or S 4 is an extra heart sound that occurs during late diastole, immediately before the normal two "lub-dub" heart sounds (S 1 and S 2).It occurs just after atrial contraction and immediately before the systolic S 1 and is caused by the atria contracting forcefully in an effort to overcome an abnormally stiff or hypertrophic ventricle.
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