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Focal neurological deficits may be caused by a variety of medical conditions such as head trauma, [1] tumors or stroke; or by various diseases such as meningitis or encephalitis or as a side effect of certain medications such as those used in anesthesia. [2] Neurological soft signs are a group of non-focal neurologic signs. [3]
The presence of symptoms defines an acute episode of functional neurologic disorder for less than six months, and persistent functional neurologic disorder includes the presence of symptoms for greater than six months. Functional neurologic disorder can also have the specifier of with or without the psychological stressor.
This is a list of major and frequently observed neurological disorders (e.g., Alzheimer's disease), symptoms (e.g., back pain), signs (e.g., aphasia) and syndromes (e.g., Aicardi syndrome). There is disagreement over the definitions and criteria used to delineate various disorders and whether some of these conditions should be classified as ...
Transient global amnesia (TGA) is a neurological disorder whose key defining characteristic is a temporary but almost total disruption of short-term memory with a range of problems accessing older memories.
Also of note is an increased risk of the development of Dementia with Lewy bodies, or (DLB), and a direct genetic association of Attention deficit disorder to Parkinson's disease [22] [23] two progressive, and serious, neurological diseases whose symptoms often occur in people over age 65. [21] [24] [25] [26]
Focal injuries typically have symptoms that are related to the damaged area of the brain. [3] Stroke can produce focal damage that is associated with signs and symptoms that correspond to the part of the brain that was damaged. [1] For example, if a speech center of the brain such as Broca's area is damaged, problems with speech are common.
Less common causes of expressive aphasia include primary autoimmune phenomenon and autoimmune phenomenon that are secondary to cancer (as a paraneoplastic syndrome) have been listed as the primary hypothesis for several cases of aphasia, especially when presenting with other psychiatric disturbances and focal neurological deficits. Many case ...
A person experiencing a TEA episode has very little short-term memory, so that there is profound difficulty remembering events in the past few minutes (anterograde amnesia), or of events in the hours before the onset of the attack, and even memories of important events in recent years may not be accessible during the amnestic event (retrograde amnesia). [6]