Search results
Results from the WOW.Com Content Network
Focal neurological deficits may be caused by a variety of medical conditions such as head trauma, [1] tumors or stroke; or by various diseases such as meningitis or encephalitis or as a side effect of certain medications such as those used in anesthesia. [2] Neurological soft signs are a group of non-focal neurologic signs. [3]
Epidemiological studies and meta-analysis have shown higher rates of depression and anxiety in patients with FND compared to the general population, but rates are similar to patients with other neurological disorders such as epilepsy or Parkinson's disease. This is often the case because of years of misdiagnosis and accusations of malingering.
Early myoclonic epileptic encephalopathy (possibly due to metabolic disorders). Gluten encephalopathy: Focal abnormalities of the white matter (generally area of low perfusion) are appreciated through magnetic resonance. Migraine is the most common symptom reported.
A ruptured microaneurysm may cause an intracerebral hemorrhage, presenting as a focal neurological deficit. [12] Rebleeding, hydrocephalus (the excessive accumulation of cerebrospinal fluid), vasospasm (spasm, or narrowing, of the blood vessels), or multiple aneurysms may also occur. The risk of rupture from a cerebral aneurysm varies according ...
Focal injuries typically have symptoms that are related to the damaged area of the brain. [3] Stroke can produce focal damage that is associated with signs and symptoms that correspond to the part of the brain that was damaged. [1] For example, if a speech center of the brain such as Broca's area is damaged, problems with speech are common.
Also of note is an increased risk of the development of Dementia with Lewy bodies, or (DLB), and a direct genetic association of Attention deficit disorder to Parkinson's disease [22] [23] two progressive, and serious, neurological diseases whose symptoms often occur in people over age 65. [21] [24] [25] [26]
Patients may have a history of loss of consciousness but they recover and do not relapse. Clinical onset occurs over hours. Complications include focal neurologic deficits depending on the site of hematoma and brain injury, increased intracranial pressure leading to herniation of brain and ischemia due to reduced blood supply and seizures.
Less common causes of expressive aphasia include primary autoimmune phenomenon and autoimmune phenomenon that are secondary to cancer (as a paraneoplastic syndrome) have been listed as the primary hypothesis for several cases of aphasia, especially when presenting with other psychiatric disturbances and focal neurological deficits. Many case ...