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Complex regional pain syndrome (CRPS type 1 and type 2), sometimes referred to by the hyponyms reflex sympathetic dystrophy (RSD) or reflex neurovascular dystrophy (RND), is a rare and severe form of neuroinflammatory and dysautonomic disorder causing chronic pain, neurovascular, and neuropathic symptoms.
This includes two separate types: type I and type II. Type I CRPS, formerly known as reflex sympathetic dystrophy (RSD) or "Sudeck's atrophy", refers to CRPS without any observed nerve damage. Type II, formerly known as causalgia, refers to CRPS with observed nerve damage. This form, similarly to other forms of AMPS, is known to be able to ...
Reflex Sympathetic Dystrophy Syndrome, the old name of the type I variation of the Complex regional pain syndrome. Reduced Swing Differential Signaling , an electronic signaling standard and protocol for a chip-to-chip interface.
Reflex sympathetic dystrophy; see complex regional pain syndrome; Rejection sensitive dysphoria in psychology; RSD may also refer to: Science
Symptoms associated with central nervous systems disorders are classified into positive and negative categories. Positive symptoms include those that increase muscle activity through hyper-excitability of the stretch reflex (i.e., rigidity and spasticity) where negative symptoms include those of insufficient muscle activity (i.e. weakness) and reduced motor function. [5]
Endoscopic thoracic sympathectomy (ETS) is a surgical procedure in which a portion of the sympathetic nerve trunk in the thoracic region is destroyed. [1] [2] ETS is used to treat excessive sweating in certain parts of the body (focal hyperhidrosis), facial flushing, Raynaud's disease and reflex sympathetic dystrophy.
Reflex Sympathetic Dystrophy, RSD, is a syndrome characterized by chronic pain, tenderness, and vasomotor instability usually in a distal extremity. Lack of recognition, familiarity and a simple diagnostic test has often delayed diagnosis and proper management.
Reflex activation then results in systemic vasoconstriction below the spinal cord disruption. This peripheral arterial vasoconstriction and hypertension activates the baroreceptors . There is a resultant parasympathetic surge originating in the central nervous system which inhibits the sympathetic outflow.