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Diatchenko also believes in and contributes to open science to advance our understanding of the molecular mechanisms of pain. Her lab's Human Pain Genetics Database (HPGdb) summarizes all extant data on the genetic contributors of pain and the Transcriptomics Pain Signatures Database (PSGS) summarizes all available data on genome-wide ...
The human Proteinpedia is based on HPRD (Human protein reference database)which is a repository hosting over 30,000 human proteins. However it is unclear how many of these are unique proteins Human Protein Atlas: The Swedish Government It contains roughly 10 million IHC images of a bit less than 25,000 antibodies.
The Single Nucleotide Polymorphism Database [1] (dbSNP) is a free public archive for genetic variation within and across different species developed and hosted by the National Center for Biotechnology Information (NCBI) in collaboration with the National Human Genome Research Institute (NHGRI).
To gather these scattered data, the Weizmann Institute of Science's Crown Human Genome Centre developed a database called ‘GeneCards’ in 1997. This database mainly dealt with human genome information, human genes, the encoded proteins’ functions, and related diseases, though it has expanded since that time.
Furthermore, Na v 1.8-null mice failed to produce action potentials, indicating that Na v 1.8 is essential to the perception of pain in cold temperatures. [19] Although the early studies on the biophysics of Na V 1.8 channels were carried out in rodent channels, more recent studies have examined the properties of human Na V 1.8 channels.
The Human Phenotype Ontology (HPO) was created in 2008 at the Charité to serve as a standardized vocabulary of phenotypic abnormalities that have been seen in human disease. Applications for the data in the ontology include clinical diagnostics, mapping between phenotypes of model organisms, and as a standard vocabulary for clinical databases.
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In humans, mutations in the PRDM12 gene can cause loss of pain perception brought on by defects in the development of sensory neurons. [8] There are a number of diseases and conditions that can result from mutations in the PRDM12 gene. Congenital insensitivity to pain (CIP) is a characterized by an inability to feel pain. [17]