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Central facial palsy is the paralysis of the lower half of one side of the face. This condition is often caused by a stroke. This condition is often the result of damage of the upper motor neurons of the facial nerve. The facial motor nucleus contains ventral and dorsal areas that have lower motor neurons that supply the upper and lower face ...
Bell's palsy is the most common cause of acute facial nerve paralysis. [3] [4] There is no known cause of Bell's palsy, [5] [6] although it has been associated with herpes simplex infection. Bell's palsy may develop over several days, and may last several months, in the majority of cases recovering spontaneously.
The facial nerve is the seventh of 12 cranial nerves. This cranial nerve controls the muscles in the face. Facial nerve palsy is more abundant in older adults than in children and is said to affect 15-40 out of 100,000 people per year. This disease comes in many forms which include congenital, infectious, traumatic, neoplastic, or idiopathic.
The facial nerve, also known as the seventh cranial nerve, cranial nerve VII, or simply CN VII, is a cranial nerve that emerges from the pons of the brainstem, controls the muscles of facial expression, and functions in the conveyance of taste sensations from the anterior two-thirds of the tongue.
Bell's palsy can trigger an increased sensitivity to sound known as hyperacusis. [6] The cause of Bell's palsy is unknown [1] and it can occur at any age. [4] Risk factors include diabetes, a recent upper respiratory tract infection, and pregnancy. [1] [7] It results from a dysfunction of cranial nerve VII (the facial nerve). [1]
The marginal mandibular branch of the facial nerve arises from the facial nerve (CN VII) in the parotid gland at the parotid plexus. It passes anterior-ward deep to the platysma and depressor anguli oris muscles.
The middle cranial foassa technique is most commonly used for the decompression of the facial nerve in Bell's palsy and longitudinal temporal bone fracture. This approach may be useful in the management of patient with schwannomas of cranial nerve 7 and 8, as well as with patient with melkersson-rosenthal syndrome.
Various kinds of tumors, usually primary and benign, are represented in the pathology. Lesions in the area of cerebellopontine angle cause signs and symptoms secondary to compression of nearby cranial nerves, including cranial nerve V (trigeminal), cranial nerve VII (facial), and cranial nerve VIII (vestibulocochlear). The most common ...