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Percutaneous coronary intervention (PCI) is a minimally invasive non-surgical procedure used to treat narrowing of the coronary arteries of the heart found in coronary artery disease. [2] The procedure is used to place and deploy coronary stents, a permanent wire-meshed tube, to open narrowed coronary arteries. PCI is considered 'non-surgical ...
This procedure makes heart surgery possible for patients who were previously considered too high risk for traditional surgery due to age or medical history. [5] [6] Patients referred for this procedure may have coronary artery disease (CAD); aortic, mitral or tricuspid valve diseases; or previous unsuccessful stenting.
A system of coaxial catheters is pushed inside the arterial circulation, usually through a percutaneous access to the right femoral artery. A microcatheter is finally positioned beyond the occluded segment and a stent-retriever is deployed to catch the thrombus; finally, the stent is pulled out from the artery, usually under continuous ...
Coronary sinus thrombosis as a severe complication after procedures. [8] The coronary sinus is the venous counterpart to the coronary arteries, where de-oxygenated blood returns from heart tissue. A large thrombus here slows overall blood circulation to heart tissue as well as may mechanically compress a coronary artery. [8]
Whenever a prosthetic device such as a valve is introduced into the bloodstream, there is an ongoing risk of thrombus formation which can lead to an embolism should the thrombus become mobile. Computational flow studies [ 21 ] conclude that the presence of an aortic valve bypass conduit has no effect on cerebral blood flow.
ATE is usually caused by atherosclerosis, which leads to plaque rupture and thrombus formation, or by cardioembolism, which results from the embolization of a cardiac thrombus, such as in atrial fibrillation, valvular disease, or myocardial dysfunction. The management of ATE depends on the location and severity of the ischemia and the ...
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Mitral stenosis causes left atrial pressure to increase, which, if left untreated, can lead to ventricular dilation, hypertrophy, atrial fibrillation, and thrombus creation. Symptoms include shortness of breath ( dyspnea ) on exertion, when lying flat ( orthopnea ) or during the night ( paroxysmal nocturnal dyspnea ), and fatigue.
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