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Periodontal disease, also known as gum disease, is a set of inflammatory conditions affecting the tissues surrounding the teeth. [5] In its early stage, called gingivitis, the gums become swollen and red and may bleed. [5] It is considered the main cause of tooth loss for adults worldwide.
Chronic periodontitis is initiated by Gram-negative tooth-associated microbial biofilms that elicit a host response, which results in bone and soft tissue destruction. In response to endotoxin derived from periodontal pathogens, several osteoclast-related mediators target the destruction of alveolar bone and supporting connective tissue such as the periodontal ligament.
Rather than a single disease entity, periodontal disease is a combination of multiple disease processes that share a common clinical manifestation. The cause includes both local and systemic factors. The disease consists of a chronic inflammation associated with loss of alveolar bone. Advanced disease features include pus and exudates.
[2] on at least two permanent teeth (one which is a first molar) and no involvement of more than two teeth other than the first molars and incisors, [2] [33] lack of inflammation and evidence of deep periodontal pocket with advanced bone loss. [2] There is also a relatively fast progression of periodontal tissue loss. [33]
Gingival recession, also known as gum recession and receding gums, is the exposure in the roots of the teeth caused by a loss of gum tissue and/or retraction of the gingival margin from the crown of the teeth. [1] Gum recession is a common problem in adults over the age of 40, but it may also occur starting in adolescence, or around the age of 10.
If attachment loss is present in the disease, it is termed NP, unless the disease has progressed beyond the mucogingival junction. NP may be an extension of NG into the periodontal ligaments, although this is not completely proven. In the meantime, NG, NP, and NS are classified together under the term necrotizing periodontal diseases. [1]
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