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  2. Etoposide - Wikipedia

    en.wikipedia.org/wiki/Etoposide

    Etoposide is a semisynthetic derivative of podophyllotoxin from the rhizome of the mayapple (or "American mandrake", Podophyllum peltatum). More specifically, it is a glycoside of podophyllotoxin with a D - glucose derivative.

  3. Topoisomerase inhibitor - Wikipedia

    en.wikipedia.org/wiki/Topoisomerase_inhibitor

    Etoposide, a semi-synthetic derivative of epipodophyllotoxin is commonly used to study this apoptotic mechanism and include: Etoposide; Teniposide; Both etoposide and teniposide are naturally occurring semi-synthetic derivatives of podophyllotoxins and are important anti-cancer drugs that function to inhibit TopII activity. [67]

  4. EPOCH (chemotherapy) - Wikipedia

    en.wikipedia.org/wiki/EPOCH_(chemotherapy)

    Etoposide: a topoisomerase inhibitor from the group of epipodophyllotoxins; Prednisolone: a glucocorticoid hormone that can cause apoptosis and lysis of both normal and malignant lymphocytes; Oncovin, also known as vincristine: a vinca alkaloid that binds to the protein tubulin, thereby preventing the formation of microtubules and mitosis;

  5. Epipodophyllotoxin - Wikipedia

    en.wikipedia.org/wiki/Epipodophyllotoxin

    Etoposide, an epipodophyllotoxin. Epipodophyllotoxins are substances naturally occurring in the root of American Mayapple plant (Podophyllum peltatum). Some epipodophyllotoxin derivatives are currently used in the treatment of cancer. These include etoposide and teniposide. They act as anti-cancer drugs by inhibiting topoisomerase II. [1]

  6. Chemotherapy - Wikipedia

    en.wikipedia.org/wiki/Chemotherapy

    It has anti-microtubule activity, and its mechanism is similar to that of vinca alkaloids in that they bind to tubulin, inhibiting microtubule formation. Podophyllotoxin is used to produce two other drugs with different mechanisms of action: etoposide and teniposide. [57] [58]

  7. Anthracycline - Wikipedia

    en.wikipedia.org/wiki/Anthracycline

    The mechanisms accounting for anthracycline-induced cardiac damage are complex and interrelated. It was first recognised to be related to the oxidative stress induced by anthracyclines. [ 29 ] A more recent explanation has emerged, in which anthracycline-mediated cardiotoxicity is due to anthracycline-topoisomerase IIb poisoning, leading to ...

  8. Mechanism of action - Wikipedia

    en.wikipedia.org/wiki/Mechanism_of_action

    A mechanism of action usually includes mention of the specific molecular targets to which the drug binds, such as an enzyme or receptor. [3] Receptor sites have specific affinities for drugs based on the chemical structure of the drug, as well as the specific action that occurs there.

  9. Teniposide - Wikipedia

    en.wikipedia.org/wiki/Teniposide

    No systematic interaction studies are available. The enzyme inducers phenobarbital and phenytoin have been found to lower its blood plasma concentrations. [4] Theoretically possible interactions include increased plasma concentrations when combined with sodium salicylate, sulfamethizole or tolbutamide, which displace teniposide from plasma protein binding, at least in vitro.

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