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Keratosis pilaris (KP; also follicular keratosis, lichen pilaris, or colloquially chicken skin. [ 1 ] ) is a common, autosomal - dominant , genetic condition of the skin's hair follicles characterized by the appearance of possibly itchy , small, gooseflesh -like bumps, with varying degrees of reddening or inflammation. [ 2 ]
Follicular hyperkeratosis, also known as keratosis pilaris (KP), is a skin condition characterized by excessive development of keratin in hair follicles, resulting in rough, cone-shaped, elevated papules. The openings are often closed with a white plug of encrusted sebum.
Keratosis (from kerat-+ -osis) [1] is a growth of keratin on the skin or on mucous membranes stemming from keratinocytes, the prominent cell type in the epidermis. More specifically, it can refer to: actinic keratosis (also known as solar keratosis), a premalignant condition; chronic scar keratosis; hydrocarbon keratosis
Keratosis pilaris atrophicans faciei begins in infancy as follicular papules with perifollicular erythema. [ 1 ] : 762 Initially, the lesions are restricted to the lateral eyebrows, but with time spread to involve the cheeks and forehead, and may also be associated with keratosis pilaris on the extremities and buttocks.
Keratosis pilaris atrophicans can be diagnosed clinically. Usually, a skin biopsy is not required to make the diagnosis. When carried out, it exhibits general characteristics such as modest perifollicular inflammatory infiltration and keratotic plugs in the pilosebaceous units.
Keratosis pilaris atrophicans faciei (folliculitis rubra, keratosis pilaris rubra atrophicans faciei, lichen pilare, lichen pilaire ou xerodermie pilaire symmetrique de la face, ulerythema ophryogenes, xerodermi pilaire symmetrique de la face) Keratosis pilaris
Keratosis pilaris is a very common genetic disorder of the hair follicle that is manifested by the appearance of rough bumps on the skin and hence colloquially referred to as "chicken skin". Photo credit: Irja from San Francisco (cc-by-sa-2.0)
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