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The treatment of equine lameness is a complex subject. Lameness in horses has a variety of causes, and treatment must be tailored to the type and degree of injury, as well as the financial capabilities of the owner. Treatment may be applied locally, systemically, or intralesionally, and the strategy for treatment may change as healing progresses.
Appropriate treatment for lameness depends on the condition diagnosed, but at a minimum it usually includes rest or decreased activity and anti-inflammatory medications. Other treatment options, such as corrective shoeing, joint injections, and regenerative therapies, are pursued based on the cause of lameness and the financial limits of the owner.
The frontal edge of the hoof cracks and splits. Pieces of hoof wall chip off in bits. This cracking, chipping and peeling may involve the whole of the dorsal hoof wall. The deficit means that when the hoof wall reaches the ground (and weightbearing load) the wall breaks away from above the level of the sole.
Regular exercise on hard or irregular ground increases concussion on the hoof, thus increasing the risk of navicular syndrome. It is possible that standing can also increase the chance of navicular disease (such as a horse that spends most of the day in a stall with little turnout, as with some racehorses and show horses).
Bone spavin indicated by A.. Bone spavin is osteoarthritis, or the final phase of degenerative joint disease (DJD), in the lower three hock joints.It usually affects the two lowest joints of the hock (the tarsometatarsal and the distal intertarsal joints), with the third joint, the proximal intertarsal, being the least likely to develop bone spavin.
Male horses are overall at a higher risk of catastrophic injury than female horses, with an overall odds ratio of 1.48. This does vary by study and country, including odds ratios of 1.12 in 1.61 in Australia, 1.76 in Canada, the United Kingdom, and 1.52–2.21 in the United States.
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