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In renal physiology, renal sodium reabsorption refers to the process by which the kidneys, having filtered out waste products from the blood to be excreted as urine, re-absorb sodium ions (Na +) from the waste. It uses Na-H antiport, Na-glucose symport, sodium ion channels (minor). [1]
The kidney is directed to excrete or retain sodium via the action of aldosterone, antidiuretic hormone (ADH, or vasopressin), atrial natriuretic peptide (ANP), and other hormones. Abnormal ranges of the fractional excretion of sodium can imply acute tubular necrosis or glomerular dysfunction.
The fractional excretion of sodium (FE Na) is the percentage of the sodium filtered by the kidney which is excreted in the urine.It is measured in terms of plasma and urine sodium, rather than by the interpretation of urinary sodium concentration alone, as urinary sodium concentrations can vary with water reabsorption.
It does so primarily by acting on the mineralocorticoid receptors in the distal tubules and collecting ducts of the nephron. [6] It influences the reabsorption of sodium and excretion of potassium (from and into the tubular fluids, respectively) of the kidney, thereby indirectly influencing water retention or loss, blood pressure, and blood ...
Sodium and potassium levels are controlled by secreting K + and absorbing Na +. Sodium absorption by the distal tubule is mediated by the hormone aldosterone. Aldosterone increases sodium reabsorption. Sodium and chloride (salt) reabsorption is also mediated by a group of kinases called WNK kinases.
ANP acts on the kidney to increase sodium and water excretion (natriuresis) in the following ways: [23] [24] The medullary collecting duct is the main site of ANP regulation of sodium excretion. [25] ANP effects sodium channels at both the apical and basolateral sides. [25]
Natriuresis is the process of sodium excretion in the urine through the action of the kidneys. [1] It is promoted by ventricular and atrial natriuretic peptides as well as calcitonin, [2] and inhibited by chemicals such as aldosterone.
Renal compensation is a mechanism by which the kidneys can regulate the plasma pH. It is slower than respiratory compensation, but has a greater ability to restore normal values. Kidneys maintain the acid-base balance through two mechanisms: (1) the secretion of H + ions into the urine (from the blood) and (2) the reabsorption of bicarbonate ...
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