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Superior alternating hemiplegia (also known as Weber syndrome) has a few distinct symptoms: contralateral hemiparesis of limb and facial muscle accompanied by weakness in one or more muscles that control eye movement on the same side. [2] Another symptom that appears is the loss of eye movement due to damage to the oculomotor nerve fibers.
Weber's syndrome, also known as midbrain stroke syndrome or superior alternating hemiplegia, is a form of stroke that affects the medial portion of the midbrain. It involves oculomotor fascicles in the interpeduncular cisterns and cerebral peduncle so it characterizes the presence of an ipsilateral lower motor neuron type oculomotor nerve palsy and contralateral hemiparesis or hemiplegia.
Impaired tactile, proprioceptive, and vibration sense of trunk and limbs (contralateral medial lemniscus) Medial medullary syndrome , also known as inferior alternating syndrome , hypoglossal alternating hemiplegia , lower alternating hemiplegia , [ 1 ] or Dejerine syndrome , [ 2 ] is a type of alternating hemiplegia characterized by a set of ...
Because of this anatomy, injuries to the pyramidal tract above the medulla generally cause contralateral hemiparesis (weakness on the opposite side as the injury). Injuries at the lower medulla, spinal cord, and peripheral nerves result in ipsilateral hemiparesis .
Contralateral spastic hemiparesis: Medial lemniscus: Contralateral PCML (aka DCML) pathway loss (tactile, vibration, and stereognosis) Abducens nerve: Strabismus (ipsilateral lateral rectus muscle paralysis - the affected eye looks down and towards the nose). Abducens nerve lesion localizes the lesion to inferior pons.
The primary motor cortex sends its axons through the posterior limb of the internal capsule. Lesions, therefore, result in a contralateral hemiparesis or hemiplegia. While symptoms of weakness due to an isolated lesion of the posterior limb can initially be severe, recovery of motor function is sometimes possible due to spinal projections of ...
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Central facial palsy is often characterized by either hemiparalysis or hemiparesis of the contralateral muscles in facial expression. [2] Muscles on the forehead are left intact. Also, most patients have lost voluntary control of muscle movement in the face—however, muscles in the face involved in spontaneous emotional expression often remain ...