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Pervasive developmental disorder: PDD-NOS Pervasive developmental disorder, not otherwise specified: PDD/NOS Pervasive developmental disorder, not otherwise specified: PDS Pokkuri Death Syndrome PE Pulmonary embolism: PKAN Panthothenate kinase-associated neurodegeneration: PLMD Periodic limb movement disorder: PLS Primary lateral sclerosis: PMD
The biology of depression is the attempt to identify a biochemical origin of depression, as opposed to theories that emphasize psychological or situational causes. Scientific studies have found that different brain areas show altered activity in humans with major depressive disorder (MDD) . [ 1 ]
In the DSM-5, it is called unspecified depressive disorder. Examples of disorders in this category include those sometimes described as minor depressive disorder and recurrent brief depression. "Depression" refers to a spectrum of disturbances in mood that vary from mild to severe and from short periods to constant illness. [1]
Endogenous depression is an atypical subclass of major depressive disorder (clinical depression). It could be caused by genetic and biological factors. [ 1 ] Endogenous depression occurs due to the presence of an internal (cognitive, biological) stressor instead of an external (social, environmental) stressor. [ 2 ]
A major depressive episode (MDE) is a period characterized by symptoms of major depressive disorder.Those affected primarily exhibit a depressive mood for at least two weeks or more, and a loss of interest or pleasure in everyday activities.
Anxiety disorders are one of the most common disorder types associated with treatment-resistant depression. The two disorders commonly co-exist, and have some similar symptoms. Some studies have shown that patients with both major depressive disorder and panic disorder are the most likely to be nonresponsive to treatment.
Depression and Anxiety is a monthly peer-reviewed medical journal published by Wiley-Blackwell.It is an official journal of the Anxiety and Depression Association of America and covers research on depressive and anxiety disorders.
For example, H3K14 and H4K12 acetylation was found to be decreased, as well as general acetylation across histones H2B and H3. [9] [10] [11] Another study found that HDAC3 was decreased in individuals resilient to depression. In the hippocampus, increased HDAC5 was found with increased depressive behavior (unlike in the nucleus accumbens). [4 ...