enow.com Web Search

Search results

  1. Results from the WOW.Com Content Network
  2. Hereditary haemochromatosis - Wikipedia

    en.wikipedia.org/wiki/Hereditary_haemochromatosis

    A study of 3,011 unrelated white Australians found that 14% were heterozygous carriers of an HFE mutation, 0.5% were homozygous for an HFE mutation, and only 0.25% of the study population had clinically relevant iron overload. Most patients who are homozygous for HFE mutations do not manifest clinically relevant haemochromatosis (see Genetics ...

  3. HFE (gene) - Wikipedia

    en.wikipedia.org/wiki/HFE_(gene)

    The protein encoded by this gene is an integral membrane protein that is similar to MHC class I-type proteins and associates with beta-2 microglobulin (beta2M). It is thought that this protein functions to regulate circulating iron uptake by regulating the interaction of the transferrin receptor with transferrin.

  4. HFE H63D gene mutation - Wikipedia

    en.wikipedia.org/wiki/HFE_H63D_gene_mutation

    This mutation is associated with diverse health issues, however H63D syndrome is the only known specific expression of a homozygous HFE-H63D mutation to date. The homozygous HFE-H63D mutation is the cause of classic and treatable hemochromatosis in only 6.7% of its carriers. [25] H63D syndrome is independently a distinct entity, and the ...

  5. Hemochromatosis type 4 - Wikipedia

    en.wikipedia.org/wiki/Hemochromatosis_type_4

    Type 4 hemochromatosis is caused by mutations of the SLC40A1 gene, located on the long arm of chromosome 2, specifically at 2q32.2. The SLC40A1 gene encodes ferroportin, a protein responsible for exporting iron from cells in the intestine, liver, spleen, and kidney, as well as from reticuloendothelial macrophages and the placenta.

  6. Methylenetetrahydrofolate reductase deficiency - Wikipedia

    en.wikipedia.org/wiki/Methylenetetrahydrofolate...

    The prevalence of the 1298C mutation is lower, at 4-12% for most tested populations. [9] A study in 2000 had identified only 24 cases of severe MTHFR deficiency (from nonsense mutations) across the whole world. [3]

  7. Nguyễn Đình Chiểu - Wikipedia

    en.wikipedia.org/wiki/Nguyễn_Đình_Chiểu

    Nguyễn Đình Chiểu was born in the southern province of Gia Định, the location of modern Saigon.He was of gentry parentage; his father was a native of Thừa Thiên–Huế, near Huế; but, during his service to the imperial government of Emperor Gia Long, he was posted south to serve under Lê Văn Duyệt, the governor of the south.

  8. Vietnamese name - Wikipedia

    en.wikipedia.org/wiki/Vietnamese_name

    TRAN LE Quoc Toan TRAN LE Q. T. Quoc Toan TRAN LE Q. T. TRAN LE [D] Bùi Hoàng Việt Anh: Bùi Hoàng: Việt Anh (no middle name) B. H. Việt Anh BUI HOANG Viet Anh BUI HOANG V. A. Viet Anh BUI HOANG V. A. BUI HOANG [E] Tôn Nữ Thị Ninh: Tôn Nữ: Thị Ninh T. N. Thị Ninh T. N. T. Ninh [B] TON NU Thi Ninh TON NU T. N. Thi Ninh TON ...

  9. Talk:HFE H63D gene mutation - Wikipedia

    en.wikipedia.org/wiki/Talk:HFE_H63D_gene_mutation

    This typo has made its way throughout the web (in research articles) and migrated into Wikipedia. The correct term is "innate immune system". 76.145.35.225 ( talk ) 15:05, 26 October 2024 (UTC) [ reply ]