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These three types of T. gondii have differing effects on certain hosts, mainly mice and humans due to their variation in genotypes. [90] Type I: virulent in mice and humans, seen in people with AIDS. Type II: non-virulent in mice, virulent in humans (mostly Europe and North America), seen in people with AIDS.
Isolated reports of scombroid food poisoning in humans caused by histamine present in the tissues of stale or rotten scombroid fish, usually tuna, have occurred over the years. In September 2016, authorities in Singapore intercepted canned tuna imported from Thailand after finding high levels of histamine. [12] No human cases were reported.
Blooms have been noted to occur as far north as North Carolina and are commonly seen alongside the widespread death of fish and sea birds. [4] In addition to the effects on human health, the economic impact of HAB-associated shellfish toxin outbreaks can have significant economic implications as well due to not only the associated healthcare ...
The threat of so-called “mad cow disease” has all but faded from the collective memory, after its appearance in U.K. cattle in 1986. Human deaths from the scourge, caused by eating ...
Amnesic shellfish poisoning (ASP) is an illness caused by consumption of shellfish that contain the marine biotoxin called domoic acid. [1] In mammals, including humans, domoic acid acts as a neurotoxin, causing permanent short-term memory loss, brain damage, and death in severe cases.
Ciguatera fish poisoning (CFP), also known as ciguatera, is a foodborne illness caused by eating reef fish contaminated with ciguatoxins. [ 4 ] [ 2 ] Such individual fish are said to be ciguatoxic . Symptoms may include diarrhea , vomiting, numbness, itchiness, sensitivity to hot and cold, dizziness , and weakness.
Ornithonyssus bacoti (also known as the tropical rat mite and formerly called Liponyssus bacoti) is a hematophagous parasite. [1] It feeds on blood and serum from many hosts. [2] [3] O. bacoti can be found and cause disease on rats and wild rodents most commonly, but also small mammals and humans when other hosts are scarce.
In the journal Immunology, Rook states that, because parasitic worms were almost always present, the human immune system developed a way to treat them that didn't cause tissue damage. [9] The immune system extends this response to its treatments of self-antigens, softening reactions against allergens, the body, [9] and digestive microorganisms ...