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There has also been research into the utility of the individual components of CBT-I, when delivered as monotherapies or multi-component therapies without cognitive therapy. A 2023 systematic review [39] demonstrated that just stimulus control and sleep restriction are effective treatment options for insomnia in older adults. It also indicated ...
Methamphetamine [note 1] (contracted from N-methylamphetamine) is a potent central nervous system (CNS) stimulant that is mainly used as a recreational or performance-enhancing drug and less commonly as a second-line treatment for attention deficit hyperactivity disorder (ADHD). [24]
Idiopathic hypersomnia (IH) is a neurological disorder which is characterized primarily by excessive sleep and excessive daytime sleepiness (EDS). [1] Idiopathic hypersomnia was first described by Bedrich Roth in 1976, and it can be divided into two forms: polysymptomatic and monosymptomatic.
Amphetamine type stimulants can be used in the treatment of narcolepsy, a rare neurological disorder where the brain is unable to regulate the sleep-wake mechanism. [17] Amphetamines causes an increase in dopamine release, which is the proposed mechanism for its wake-promoting effect. [ 18 ]
Atypical and typical antipsychotics have been shown to be helpful in the early stages of treatment, especially olanzapine over haloperidol. [8] The benzodiazepines temazepam and triazolam at 30 mg and 0.5 mg (prescribed independently from olanzapine [ 39 ] [ 40 ] and haloperidol [ 41 ] [ 42 ] ), are highly effective if aggression, agitation, or ...
Between 10% and 30% of adults have insomnia at any given point in time and up to half of people have insomnia in a given year, making it the most common sleep disorder. [ 9 ] [ 8 ] [ 10 ] [ 207 ] About 6% of people have insomnia that is not due to another problem and lasts for more than a month. [ 9 ]
Under the developmental code names EMP-01 and MM-402, it is under development for the treatment of post-traumatic stress disorder (PTSD), social phobia, and pervasive development disorders (PDDs) such as autism. [7] [8] [9] It is thought that (R)-MDMA might have a better safety profile than MDMA itself whilst retaining its therapeutic benefits. [3]
Administration of MDMA to mice causes DNA damage in their brain, [83] especially when the mice are sleep deprived. [84] Even at the very low doses that are comparable to those self-administered by humans, MDMA causes oxidative stress and both single and double-strand breaks in the DNA of the hippocampus region of the mouse brain.
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