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Diagnosis of neurogenic claudication is based on typical clinical features, the physical exam, and findings of spinal stenosis on computer tomography (CT) or X-ray imaging. [1] In addition to vascular claudication, diseases affecting the spine and musculoskeletal system should be considered in the differential diagnosis. [9]
Claudication is most common in the calves but it can also affect the feet, thighs, hips, buttocks, or arms. [2] The word claudication comes from Latin claudicare 'to limp'. Claudication that appears after a short amount of walking may sometimes be described by US medical professionals by the number of typical city street blocks that the patient ...
However, a retromalleolar hypovascular region exists in the area and may contribute to the disease. When autopsied, cadavers with the disease show decreased blood supply. The position of the tendon is also thought to contribute, as it makes a sharp turn around the medial malleolus , putting a lot of tension on the tendon .
This results in claudication and chronic leg ischemia. This condition mainly occurs more in young athletes than in the elderlies. [2] Elderlies, who present with similar symptoms, are more likely to be diagnosed with peripheral artery disease with associated atherosclerosis. [2]
Peripheral artery disease is typically diagnosed if the systolic blood pressure in the ankle divided by the systolic blood pressure in the arm is less than 0.9. In general, claudication is often seen in people with an ABI between 0.4 and 0.9, rest pain is seen between 0.2 and 0.4, and tissue loss, ulcers, and gangrene between 0 to 0.4.
The most common symptoms of the disease "are balance and walking difficulties, clumsiness, vision changes, speech difficulties, ... "but poor sensation in the feet, inner-ear problems or vision ...
Intermittent claudication is a symptom and is by definition diagnosed by a patient reporting a history of leg pain with walking relieved by rest. However, as other conditions such as sciatica can mimic intermittent claudication, testing is often performed to confirm the diagnosis of peripheral artery disease. [citation needed]
The disease mechanism (pathophysiology) of RS3PE remains unknown. One study suggested a possible role for vascular endothelial growth factor. [13] A study using magnetic resonance imaging found that tenosynovitis of the extensors of the hands and feet is the major contributor to edema. [14]