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[citation needed] This reflex may also be impaired by damage to the internal branch of the superior laryngeal nerve which relays the afferent branch of the reflex arc. [ citation needed ] This nerve is most commonly damaged by swallowing a foreign object, such as a chicken bone, resulting in it being lodged in the piriform recess (in the ...
Contact granuloma is a condition that develops due to persistent tissue irritation in the posterior larynx. [1] [2] Benign granulomas, not to be confused with other types of granulomas, occur on the vocal process of the vocal folds, where the vocal ligament attaches.
However, on the other end of the spectrum are people with a hypersensitive gag reflex. This hypersensitivity can lead to issues in various situations, such as swallowing a pill or large bites of food, or visiting the dentist. Hypersensitivity is generally a conditioned response, usually occurring following a previous experience.
Vocal cord paresis, also known as recurrent laryngeal nerve paralysis or vocal fold paralysis, is an injury to one or both recurrent laryngeal nerves (RLNs), which control all intrinsic muscles of the larynx except for the cricothyroid muscle. The RLN is important for speaking, breathing and swallowing.
Laryngopharyngeal reflux (LPR) or laryngopharyngeal reflux disease (LPRD) is the retrograde flow of gastric contents into the larynx, oropharynx and/or the nasopharynx. [4] [5] LPR causes respiratory symptoms such as cough and wheezing [6] and is often associated with head and neck complaints such as dysphonia, globus pharyngis, and dysphagia. [7]
Serum sickness is a type III hypersensitivity reaction, caused by immune complexes. [2] When an antiserum is given, the human immune system can mistake the proteins present for harmful antigens . The body produces antibodies, which combine with these proteins to form immune complexes. [ 2 ]
Hypersensitivity Pneumonitis; Other names: Allergic alveolitis, bagpipe lung, extrinsic allergic alveolitis (EAA) High magnification photomicrograph of a lung biopsy taken showing chronic hypersensitivity pneumonitis (), showing mild thickening of the walls of the small air sacs by invasion of white blood cells.
Later, it became clear that several different disease mechanisms were implicated, with a common link to a disordered activation of the immune system. In 1963, a new classification scheme was designed by Philip Gell and Robin Coombs that described four types of hypersensitivity reactions, known as Type I to Type IV hypersensitivity. [152]