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Strontium ranelate is an antiosteoporotic agent which both increases bone formation and reduces bone resorption, resulting in a rebalance of bone turnover in favor of bone formation. This is similar to the effects of choline-stabilized orthosilicic acid.
The use of strontium ranelate is restricted because of increased risk of venous thromboembolism, pulmonary embolism and serious cardiovascular disorders, including myocardial infarction. [8] In postmenopausal women, the selective estrogen receptor modulator raloxifene is occasionally administered instead of bisphosphonates.
Its beneficial effects are also questionable, since the increased bone density is partially caused by the increased density of strontium over the calcium which it replaces. Strontium also bioaccumulates in the body. [97] Despite restrictions on strontium ranelate, strontium is still contained in some supplements.
It forms the ranelate ion, C 12 H 6 N 2 O 8 S 4−. Strontium ranelate , the strontium salt of ranelic acid, is a drug used to treat osteoporosis and increase bone mineral density (BMD). References
[2] [4] Stable bone seekers can also be harmful: excessive strontium absorption has been linked with increased levels of rickets. [5] [6] The salt strontium ranelate, however, is a bone seeker which is sometimes used to strengthen bones as a treatment for osteoporosis. [7]
Allopurinol and sulfasalazine account for almost 66% of DRESS syndrome cases with minocycline being the third most common cause of the disorder; Strontium ranelate, leflunomide, dapsone, and nonsteroidal anti-inflammatory drugs (diclofenac, celecoxib, ibuprofen, and phenylbutazone) are less common causes of the disorder. [10]
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Strontium ranelate – may decrease degeneration in osteoarthritis and improve outcomes [202] [203] Gene therapy – Gene transfer strategies aim to target the disease process rather than the symptoms. [204] Cell-mediated gene therapy is also being studied.
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