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  2. Neuromuscular-blocking drug - Wikipedia

    en.wikipedia.org/wiki/Neuromuscular-blocking_drug

    IV dose 1-1.5mg/kg or 3 to 5 x ED 95. Paralysis occurs in one to two minutes. Clinical duration of action (time from drug administration to recovery of single twich to 25% of baseline) is 7-12 minutes. If IV access is unavailable, intramuscular administration 3-4mg/kg. Paralysis occurs at 4 minutes.

  3. Anticholinergic - Wikipedia

    en.wikipedia.org/wiki/Anticholinergic

    [4] The term "anticholinergic" is typically used to refer to antimuscarinics that competitively inhibit the binding of ACh to muscarinic acetylcholine receptors; such agents do not antagonize the binding at nicotinic acetylcholine receptors at the neuromuscular junction, although the term is sometimes used to refer to agents that do so. [3] [5]

  4. Cholinergic blocking drug - Wikipedia

    en.wikipedia.org/wiki/Cholinergic_blocking_drug

    [1] [3] [4] According to their site of actions, cholinergic blocking drugs can be classified into two general types — antimuscarinic and antinicotinic agents. [1] Antimuscarinic agents (also known as muscarinic antagonists), including atropine and hyoscine, block acetylcholine at the muscarinic acetylcholine receptors.

  5. Leupeptin - Wikipedia

    en.wikipedia.org/wiki/Leupeptin

    Leupeptin inhibits serine proteinases (trypsin (K i =3.5 nM), plasmin (K i = 3.4 nM), porcine kallikrein), and cysteine proteinases (papain, cathepsin B (K i = 4.1 nM), endoproteinase Lys-C). It does not inhibit α-chymotrypsin or thrombin. Leupeptin is a competitive transition state inhibitor and its inhibition may be relieved by an excess of ...

  6. Neuromuscular drug - Wikipedia

    en.wikipedia.org/wiki/Neuromuscular_drug

    Acetylcholinesterase inhibitors: Acetylcholinesterase inhibitors prevent the degradation of acetylcholine, subsequently increasing its concentration and duration of action in the neuromuscular junction. [8] Acetylcholinesterase inhibitors are primarily used to treat symptoms of dementia, Alzheimer's disease, Parkinson's disease, and myasthenia ...

  7. Nav1.8 - Wikipedia

    en.wikipedia.org/wiki/Nav1.8

    Na v 1.7, Na v 1.8 and Na v 1.9 are found in the DRG and help mediate chronic inflammatory pain. [13] Na v 1.8 is an α-type channel subunit consisting of four homologous domains, each with six transmembrane regions, of which one is a voltage sensor. Structure of Na v 1.8, an α-type subunit with four homologous domains, each with six ...

  8. Receptor antagonist - Wikipedia

    en.wikipedia.org/wiki/Receptor_antagonist

    Antagonists will block the binding of an agonist at a receptor molecule, inhibiting the signal produced by a receptor–agonist coupling.. A receptor antagonist is a type of receptor ligand or drug that blocks or dampens a biological response by binding to and blocking a receptor rather than activating it like an agonist.

  9. Metalloprotease inhibitor - Wikipedia

    en.wikipedia.org/wiki/Metalloprotease_inhibitor

    These inhibitors bind through two phosphonate oxygen atoms. Phosphonate inhibitors have been developed that exhibit selectivity for MMP-8 over other MMPs. Selective MMP-8 inhibitors could be useful in the treatment of acute liver disease and multiple sclerosis [15] Phosphinic MMP inhibitors have been reported to target MMP-11 and MMP-13.