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Atrial tachycardia is a type of heart rhythm problem in which the heart's electrical impulse comes from an ectopic pacemaker (that is, an abnormally located cardiac pacemaker) in the upper chambers of the heart, rather than from the sinoatrial node, the normal origin of the heart's electrical activity.
An upsloping, convex ST segment is highly predictive of a myocardial infarction (Pardee sign) while a concave ST elevation is less suggestive and can be found in other non-ischaemic causes. [1] Following infarction, ventricular aneurysm can develop, which leads to persistent ST elevation, loss of S wave, and T wave inversion. [1]
Acute coronary syndrome is subdivided in three scenarios depending primarily on the presence of electrocardiogram (ECG) changes and blood test results (a change in cardiac biomarkers such as troponin levels): [4] ST elevation myocardial infarction (STEMI), non-ST elevation myocardial infarction (NSTEMI), or unstable angina. [5]
Ectopic atrial tachycardia (EAT), also known as automatic atrial tachycardia, is an arrhythmia caused by both atria with abnormally fast atrial rates. [12] The ectopic focus's firing rate is quicker than that of the sinus node, and it overrides normal sinus node activity. Heart rates in children and adolescents can range from 130 to 210 beats ...
Atrial tachycardia with complete A-V block and resulting junctional escape. Many conditions can cause third-degree heart block, but the most common cause is coronary ischemia. Progressive degeneration of the electrical conduction system of the heart can lead to third-degree heart block.
An ECG, which is a recording of the heart's electrical activity, may confirm an ST elevation MI , if ST elevation is present. [8] [15] Commonly used blood tests include troponin and less often creatine kinase MB. [7] Treatment of an MI is time-critical. [16] Aspirin is an appropriate immediate treatment for a suspected MI. [9]
Typically, the atrial impulse propagates normally through the atrioventricular node and into the cardiac ventricles, resulting in a normal, narrow QRS complex. However, if the atrial beat is premature enough, it may reach the atrioventricular node during its refractory period, in which case it will not be conducted to the ventricle and there ...
ST-T Abnormalities: ST-T vector opposite to QRS without digitalis; ST-T vector opposite to QRS with digitalis; 3 1 Negative terminal P mode in V 1 1 mm in depth and 0.04 sec in duration (indicates left atrial enlargement) 3 Left axis deviation (QRS of −30° or more) 2 QRS duration ≥0.09 sec 1 Delayed intrinsicoid deflection in V 5 or V 6 ...
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