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  2. Agonist - Wikipedia

    en.wikipedia.org/wiki/Agonist

    One example of a drug that can act as a full agonist is isoproterenol, which mimics the action of adrenaline at β adrenoreceptors. Another example is morphine, which mimics the actions of endorphins at μ-opioid receptors throughout the central nervous system. However, a drug can act as a full agonist in some tissues and as a partial agonist ...

  3. 5-HT2C receptor agonist - Wikipedia

    en.wikipedia.org/wiki/5-HT2c_receptor_agonist

    Vabicaserin is a full agonist with approximately 4-fold greater selectivity for 5-HT 2C over these related receptors, in terms of binding affinity. Vabicacserin is a full agonist in stimulating the 5-HT 2C receptor; it was discovered when a class of tetrahydroquinoline-fused diazepines were being researched as possible potent 5-HT 2C receptor ...

  4. Drug action - Wikipedia

    en.wikipedia.org/wiki/Drug_action

    Receptors are located on all cells in the body. The same receptor can be located on different organs, and even on different types of tissues. There are also different subtypes of receptor which elicit different effects in response to the same agonist. For example, there are two types of histamine receptor: H1 and H2. Activation of the H1 ...

  5. Beta2-adrenergic agonist - Wikipedia

    en.wikipedia.org/wiki/Beta2-adrenergic_agonist

    Beta 2-adrenergic agonists, also known as adrenergic β 2 receptor agonists, are a class of drugs that act on the β 2 adrenergic receptor. Like other β adrenergic agonists , they cause smooth muscle relaxation. β 2 adrenergic agonists' effects on smooth muscle cause dilation of bronchial passages , vasodilation in muscle and liver ...

  6. Allosteric modulator - Wikipedia

    en.wikipedia.org/wiki/Allosteric_modulator

    Modulators that increase only the affinity of partial and full agonists allow their efficacy maximum to be reached sooner at lower agonist concentrations – i.e. the slope and plateau of a dose-response curve shift to lower concentrations. [4] Efficacy increasing modulators increase maximum efficacy of partial agonists.

  7. Receptor (biochemistry) - Wikipedia

    en.wikipedia.org/wiki/Receptor_(biochemistry)

    Partial agonists do not activate receptors with maximal efficacy, even with maximal binding, causing partial responses compared to those of full agonists (efficacy between 0 and 100%). Antagonists bind to receptors but do not activate them. This results in a receptor blockade, inhibiting the binding of agonists and inverse agonists.

  8. Dopamine agonist - Wikipedia

    en.wikipedia.org/wiki/Dopamine_agonist

    Dopamine agonists act directly on the dopamine receptors and mimic dopamine's effect. [1] Dopamine agonists have two subclasses: ergoline and non-ergoline agonists. Both subclasses target dopamine D 2-type receptors. Types of ergoline agonists are cabergoline and bromocriptine and examples of non-ergoline agonists are pramipexole, ropinirole ...

  9. Pharmacology - Wikipedia

    en.wikipedia.org/wiki/Pharmacology

    Ligands can be agonists, partial agonists or antagonists at specific receptors in the body. Agonists bind to receptors and produce a biological response, a partial agonist produces a biological response lower than that of a full agonist, antagonists have affinity for a receptor but do not produce a biological response.